期刊
STROKE
卷 38, 期 2, 页码 753-758出版社
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1161/01.STR.0000255033.02904.db
关键词
blood-brain barrier; edema, brain; free radicals; inflammation; intracranial hemorrhage; treatment
资金
- NINDS NIH HHS [R01NS-30652] Funding Source: Medline
A significant amount of new information has been generated in animal models of intracerebral hemorrhage during the past several years. These include findings on the pathophysiological, biochemical and molecular processes that underlie the development of brain tissue injury after intracerebral hemorrhage as well as potential new treatments. We review these various findings that include glutamate receptor activation, oxidative stress development, intracellular signaling through the transcription factor, nuclear factor-kappa B, and markedly upregulated cytokine gene expression. We also briefly review the surgical treatment for intracerebral hemorrhage and list the pharmacological treatment studies that have recently appeared. (Stroke. 2007;38[part 2]:753-758.)
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据