期刊
TRENDS IN GENETICS
卷 23, 期 10, 页码 475-477出版社
ELSEVIER SCIENCE LONDON
DOI: 10.1016/j.tig.2007.08.008
关键词
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Genetic studies in several species have demonstrated that myostatin (MSTN) normally functions to limit skeletal muscle mass. In a recent study, Mosher et al. reported that a mutation in the canine MSTN gene is responsible for the double-muscling phenotype seen in 'bully' whippets. Furthermore, they show that loss of even one functional MSTN allele seems to confer a competitive advantage to racing whippets, providing the first definitive evidence that loss of myostatin function can enhance athletic performance.
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