期刊
ADVANCED MATERIALS
卷 19, 期 19, 页码 2790-+出版社
WILEY-V C H VERLAG GMBH
DOI: 10.1002/adma.200602696
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The biomolecular mechanism of nickel carcinogenicity is driven by intracellular Ni cation. The role of nickel catalyst in single-wall carbon nanotube toxicity will therefore depend on its bioavailability, which is highly uncertain due to encapsulation by carbon shells. This article measures the material-specific Ni release into extra- and intracellular physiological fluid phases and suggests practical techniques for managing the metals contribution to SWNT toxicity.
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