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M. Okamoto et al.
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Bleomycin and IL-1β-mediated pulmonary fibrosis is IL-17A dependent
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Periostin Associates with Notch1 Precursor to Maintain Notch1 Expression under a Stress Condition in Mouse Cells
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The multifaceted role of periostin in tumorigenesis
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Genetic Manipulation of Periostin Expression in the Heart Does Not Affect Myocyte Content, Cell Cycle Activity, or Cardiac Repair
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The many facets of the matricelluar protein periostin during cardiac development, remodeling, and pathophysiology
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Murine models of pulmonary fibrosis
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Periostin is required for maturation and extracellular matrix stabilization of noncardiomyocyte lineages of the heart
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Periostin, secreted from stromal cells, has biphasic effect on cell migration and correlates with the epithelial to mesenchymal transition of human pancreatic cancer cells
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Periostin is essential for cardiac healing after acute myocardial infarction
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Periostin facilitates eosinophil tissue infiltration in allergic lung and esophageal responses
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Functional role of periostin in development and wound repair: implications for connective tissue disease
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Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling
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Angiogenesis inhibition as a therapeutic approach for inflammatory synovitis
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Periostin induces proliferation of differentiated cardiomyocytes and promotes cardiac repair
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Mice lacking neutrophil elastase are resistant to bleomycin-induced pulmonary fibrosis
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Periostin: A novel component of subepithelial fibrosis of bronchial asthma downstream of IL-4 and IL-13 signals
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Predictive factors for interstitial lung disease, antitumor response, and survival in non-small-cell lung cancer patients treated with gefitinib
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JOURNAL OF CLINICAL ONCOLOGY (2006)
periostin null mice exhibit dwarfism, incisor enamel defects, and an early-onset periodontal disease-like phenotype
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MOLECULAR AND CELLULAR BIOLOGY (2005)
Pathogenesis and natural history of usual interstitial pneumonia - The whole story or the last chapter of a long novel
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Bleomycin-induced E prostanoid receptor changes alter fibroblast responses to prostaglandin E2
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C-C chemokine receptor 2 (CCR2) deficiency improves bleomycin-induced pulmonary fibrosis by attenuation of both macrophage infiltration and production of macrophage-derived matrix metalloproteinases
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