相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Knockout of the Na,K-ATPase α2-isoform in the cardiovascular system does not alter basal blood pressure but prevents ACTH-induced hypertension
Tara N. Rindler et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2011)
Mice expressing ouabain-sensitive alpha 1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy
Arshani N. Wansapura et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2011)
Hypertension from chronic central sodium chloride in mice is mediated by the ouabain-binding site on the Na,K-ATPase α2-isoform
James W. Van Huysse et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2011)
The central role of the brain aldosterone-ouabain pathway in salt-sensitive hypertension
Frans H. H. Leenen
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2010)
Molecular and Functional Characterization of a Novel Cardiac-Specific Human Tropomyosin Isoform
Sudarsan Rajan et al.
CIRCULATION (2010)
Extracellular potassium dependence of the Na+-K+-ATPase in cardiac myocytes: isoform specificity and effect of phospholemman
Fei Han et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2009)
Enhanced pressor response to increased CSF sodium concentration and to central ANG I in heterozygous α2 Na+-K+-ATPase knockout mice
Xiaohong Hou et al.
AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY (2009)
ACTH-induced hypertension is dependent on the ouabain-binding site of the α2-Na+-K+-ATPase subunit
John N. Lorenz et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2008)
Differential distribution and regulation of mouse cardiac Na+/K+-ATPase α1 and α2 subunits in T-tubule and surface sarcolemmal membranes
Roger G. Berry et al.
CARDIOVASCULAR RESEARCH (2007)
Functional characterization of Src-interacting Na/K-ATPase using RNA interference assay
Man Liang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Cardiac-specific deletion of Gata4 reveals its requirement for hypertrophy, compensation, and myocyte viability
T Oka et al.
CIRCULATION RESEARCH (2006)
Physiological role of the α1- and α2-isoforms of the Na+-K+-ATPase and biological significance of their cardiac glycoside binding site
I Dostanic-Larson et al.
AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY (2006)
Sodium pump α2 subunits control myogenic tone and blood pressure in mice
J Zhang et al.
JOURNAL OF PHYSIOLOGY-LONDON (2005)
The highly conserved cardiac glycoside binding site of Na,K-ATPase plays a role in blood pressure regulation
I Dostanic-Larson et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
The α2-isoform of Na-K-ATPase mediates ouabain-induced hypertension in mice and increased vascular contractility in vitro
I Dostanic et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2005)
Na+ pump α2-isoform specifically couples to contractility in vascular smooth muscle:: evidence from gene-targeted neonatal mice
DA Shelly et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2004)
The α1 isoform of Na,K-ATPase regulates cardiac contractility and functionally interacts and co-localizes with the Na/Ca exchanger in heart
I Dostanic et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Genetic loss of calcineurin blocks mechanical overload-induced skeletal muscle fiber type switching but not hypertrophy
SA Parsons et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
The α2 isoform of Na,K-ATPase mediates ouabain-induced cardiac inotropy in mice
I Dostanic et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Na+/K+-ATPase as a signal transducer
ZJ Xie et al.
EUROPEAN JOURNAL OF BIOCHEMISTRY (2002)
Telemetric monitoring of adrenocorticotrophin-induced hypertension in mice
CG Schyvens et al.
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY (2001)
High salt intake and the brain renin-angiotensin system in Dahl salt-sensitive rats
XG Zhao et al.
JOURNAL OF HYPERTENSION (2001)
分享论文
