相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Cardiac-specific overexpression of the human type 1 angiotensin II receptor causes delayed repolarization
Katy Rivard et al.
CARDIOVASCULAR RESEARCH (2008)
Postnatal development has a marked effect on ventricular repolarization in mice
Scott A. Grandy et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
The acid-sensitive potassium channel TASK-1 in rat cardiac muscle
Caroline Putzke et al.
CARDIOVASCULAR RESEARCH (2007)
Targeted deletion of Kv4.2 eliminates Ito,f and results in electrical and molecular remodeling, with no evidence of ventricular hypertrophy or myocardial dysfunction
WN Guo et al.
CIRCULATION RESEARCH (2005)
Both α1A- and α1B-adrenergic receptors crosstalk to downregulate β1-ARs in mouse heart:: coupling to differential PTX-sensitive pathways
BR Rorabaugh et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2005)
Isolation and characterization of subcellular protein fractions from mouse heart
E Lizotte et al.
ANALYTICAL BIOCHEMISTRY (2005)
Molecular and functional signature of heart hypertrophy during pregnancy
M Eghbali et al.
CIRCULATION RESEARCH (2005)
Sex and strain differences in adult mouse cardiac repolarization: importance of androgens
J Brouillette et al.
CARDIOVASCULAR RESEARCH (2005)
Functional properties of K+ currents in adult mouse ventricular myocytes
J Brouillette et al.
JOURNAL OF PHYSIOLOGY-LONDON (2004)
In vivo cardiac gene transfer of Kv4.3 abrogates the hypertrophic response in rats after aortic stenosis
D Lebeche et al.
CIRCULATION (2004)
A fundamental role for KChIPs in determining the molecular properties and trafficking of Kv4.2 potassium channels
R Shibata et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Properties, expression and potential roles of cardiac K+ channel accessory subunits:: MinK, MiRPs, KChIP, and KChAP
M Pourrier et al.
JOURNAL OF MEMBRANE BIOLOGY (2003)
The long QT interval is not only inherited but is also linked to cardiac hypertrophy
B Swynghedauw et al.
JOURNAL OF MOLECULAR MEDICINE-JMM (2003)
Effect of androgen deficiency on mouse ventricular repolarization
J Brouillette et al.
JOURNAL OF PHYSIOLOGY-LONDON (2003)
Cardiovascular influences of α1b-adrenergic receptor defect in mice
C Vecchione et al.
CIRCULATION (2002)
Reduction of Ito causes hypertrophy in neonatal rat ventricular myocytes
Z Kassiri et al.
CIRCULATION RESEARCH (2002)
A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of Ito and confers susceptibility to ventricular tachycardia
HC Kuo et al.
CELL (2001)
Mechanism of α-adrenergic regulation of expressed hKv4.3 currents
SS Po et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2001)
Angiotensin II inhibits and alters kinetics of voltage-gated K+ channels of rat arterial smooth muscle
Y Hayabuchi et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2001)
Gender-based differences in cardiac repolarization in mouse ventricle
W Trépanier-Boulay et al.
CIRCULATION RESEARCH (2001)
The consequences of disrupting cardiac inwardly rectifying K+ current (IK1) as revealed by the targeted deletion of the murine Kir2.1 and Kir2.2 genes
JJ Zaritsky et al.
JOURNAL OF PHYSIOLOGY-LONDON (2001)
Targeted replacement of Kv1.5 in the mouse leads to loss of the 4-aminopyridine-sensitive component of IK,slow and resistance to drug-induced QT prolongation
B London et al.
CIRCULATION RESEARCH (2001)
Independent regulation of cardiac Kv4.3 potassium channel expression by angiotensin II and phenylephrine
TT Zhang et al.
CIRCULATION RESEARCH (2001)