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Postconditioning reduces infarct size via adenosine receptor activation by endogenous adenosine
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Mechanism of cGMP-mediated protection in a cellular model of myocardial reperfusion injury
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Ischemic preconditioning protects by activating prosurvival kinases at reperfusion
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Postconditioning's protection is not dependent on circulatin blood factors or cells but involves adenosine receptors and requires PI3-kinase and guanylyl cyclase activation
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Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways
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Exogenous NO triggers preconditioning via a cGMP- and mitoKATP-dependent mechanism
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ATP-sensitive K+ channel activation by nitric oxide and protein kinase G in rabbit ventricular myocytes
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AMP 579 reduces contracture and limits infarction in rabbit heart by activating adenosine A2 receptors
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Acetylcholine, bradykinin, opioids, and phenylephrine, but not adenosine, trigger preconditioning by generating free radicals and opening mitochondrial KATP channels
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