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注意:仅列出部分参考文献,下载原文获取全部文献信息。The WNKs: Atypical Protein Kinases With Pleiotropic Actions
James A. McCormick et al.
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Epigenetic silencing of the kinase tumor suppressor WNK2 is tumor-type and tumor-grade specific
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Autocrine factors that sustain glioma invasion and paracrine biology in the brain microenvironment
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The thiazide-sensitive Na-Cl cotransporter is regulated by a WNK kinase signaling complex
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Protein kinase WNK2 inhibits cell proliferation by negatively modulating the activation of MEK1/ERK1/2
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Amiloride-sensitive Na+ channels contribute to regulatory volume increases in human glioma cells
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Epigenome scans and cancer genome sequencing converge on WNK2, a kinase-independent suppressor of cell growth
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Extracellular glutamine is a critical modulator for regulatory volume increase in human glioma cells
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A single binding motif is required for SPAK activation of the Na-K-2Cl cotransporter
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WNK1 and OSR1 regulate the Na+, K+, 2Cl- cotransporter in HeLa cells
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Protein kinase WNK3 increases cell survival in a caspase-3-dependent pathway
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Volume sensitivity of cation-Cl- cotransporters is modulated by the interaction of two kinases: Ste20-related proline-alanine-rich kinase and WNK4
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Properties of WNK1 and implications for other family members
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Relative contribution of chloride channels and transporters to regulatory volume decrease in human glioma cells
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W Chen et al.
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Ion channels and amino acid transporters support the growth and invasion of primary brain tumors
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Cloning, genomic organization, alternative splicing and expression analysis of the human gene RWK3 (PRKWNK3)
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Characterization of the interaction of the stress kinase SPAK with the Na+-K+-2Cl- cotransporter in the nervous system -: Evidence for a scaffolding role of the kinase
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Modulation of ion transport by direct targeting of protein phosphatase type 1 to the Na-K-Cl cotransporter
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