相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Role of HIF-1α and VEGF in human mesenchymal stem cell proliferation by 17β-estradiol: involvement of PKC, PI3K/Akt, and MAPKs
Seung Pil Yun et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2009)
Arachidonic acid potentiates hypoxia-induced VEGF expression in mouse embryonic stem cells: involvement of Notch, Wnt, and HIF-1α
Sang Hun Lee et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2009)
Focal adhesion kinase signaling pathway participates in the formation of choroidal neovascularization and regulates the proliferation and migration of choroidal microvascular endothelial cells by acting through HIF-1 and VEGF expression in RPE cells
Jie Zhu et al.
EXPERIMENTAL EYE RESEARCH (2009)
Hypoxia or In Situ Normoxia: The Stem Coll Paradigm
Zoran Ivanovic
JOURNAL OF CELLULAR PHYSIOLOGY (2009)
HB-EGF-induced VEGF production and eNOS activation depend on both PI3 kinase and MAP kinase in HaCaT cells
Kozo Nakai et al.
JOURNAL OF DERMATOLOGICAL SCIENCE (2009)
Short-period hypoxia increases mouse embryonic stem cell proliferation through cooperation of arachidonic acid and PI3K/Akt signalling pathways
S. H. Lee et al.
CELL PROLIFERATION (2008)
Hypoxic preconditioning results in increased motility and improved therapeutic potential of human mesenchymal stem cells
Ivana Rosova et al.
STEM CELLS (2008)
Acetylcholine inhibits long-term hypoxia-induced apoptosis by suppressing the oxidative stress-mediated MAPKs activation as well as regulation of Bcl-2, c-IAPs, and caspase-3 in mouse embryonic stem cells
Min Hee Kim et al.
APOPTOSIS (2008)
The hypoxia-inducible factor HIF-1 promotes intramyocardial expression of VEGF in infants with congenital cardiac defects
Ma Quing et al.
BASIC RESEARCH IN CARDIOLOGY (2007)
Transcriptional profiling of human cord blood CD133(+) and cultured bone marrow mesenchymal stem cells in response to hypoxia
Enca Martin-Rendon et al.
STEM CELLS (2007)
New concepts regarding focal promotion of cell migration adhesion kinase and proliferation
Braden D. Cox et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2006)
Short time exposure to hypoxia promotes H9c2 cell growth
Rie Takahashi et al.
BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS (2006)
Vascular endothelial growth factor receptor-1 mediates migration of human colorectal carcinoma cells by activation of Src family kinases
D. P. Lesslie et al.
BRITISH JOURNAL OF CANCER (2006)
Expression of vascular endothelial growth factor receptor 1 in bone marrow-derived mesenchymal cells is dependent on hypoxia-inducible factor 1
Hiroaki Okuyama et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Focal adhesions - Paradigm for a signaling nexus
LH Romer et al.
CIRCULATION RESEARCH (2006)
Soluble vascular endothelial growth factor receptor-1 (sFLT-1) mediates downregulation of FLT-1 and prevents activated neutrophils from women with preeclampsia from additional migration by VEGF
O Krysiak et al.
CIRCULATION RESEARCH (2005)
VEGF-A and PlGF-1 stimulate chemotactic migration of human mesenchymal progenitor cells
J Fiedler et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2005)
Purine nucleoside-mediated protection of chemical hypoxia-induced neuronal injuries involves p42/44 MAPK activation
B Tomaselli et al.
NEUROCHEMISTRY INTERNATIONAL (2005)
Low O2 tensions and the prevention of differentiation of hES cells
T Ezashi et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Postnatal bone marrow stromal cells elicit a potent VEGF-dependent neoangiogenic response in vivo
A Al-Khaldi et al.
GENE THERAPY (2003)
Hypoxia promotes murine bone-marrow-derived stromal cell migration and tube formation
B Annabi et al.
STEM CELLS (2003)
Vascular endothelial growth factor and angiopoietin-1 stimulate postnatal hematopoiesis by recruitment of vasculogenic and hematopoietic stem cells
K Hattori et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2001)
Hypoxia-inducible factor and the development of stem cells of the cardiovascular system
DL Ramirez-Bergeron et al.
STEM CELLS (2001)
Vascular endothelial growth factor (VEGF)-driven actin-based motility is mediated by VEGFR2 and requires concerted activation of stress-activated protein kinase 2 (SAPK2/p38) and geldanamycin-sensitive phosphorylation of focal adhesion kinase
S Rousseau et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
分享论文
