4.7 Article

Intrinsic apoptotic pathway activation increases response to anti-estrogens in luminal breast cancers

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Biochemical Research Methods

Visualization and quantification of protein-protein interactions in cells and tissues

Mats Gullberg et al.

NATURE METHODS (2018)

Article Biochemistry & Molecular Biology

Discovery and biological characterization of potent myeloid cell leukemia-1 inhibitors

Taekyu Lee et al.

FEBS LETTERS (2017)

Article Oncology

Key Survival Factor, Mcl-1, Correlates with Sensitivity to Combined Bcl-2/Bcl-xL Blockade

Michelle M. Williams et al.

MOLECULAR CANCER RESEARCH (2017)

Article Multidisciplinary Sciences

The MCL1 inhibitor S63845 is tolerable and effective in diverse cancer models

Andras Kotschy et al.

NATURE (2016)

Article Cell Biology

PIK3CA mutations enable targeting of a breast tumor dependency through mTOR-mediated MCL-1 translation

Grace R. Anderson et al.

SCIENCE TRANSLATIONAL MEDICINE (2016)

Article Multidisciplinary Sciences

Assessment of ABT-263 activity across a cancer cell line collection leads to a potent combination therapy for small-cell lung cancer

Anthony C. Faber et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2015)

Article Multidisciplinary Sciences

Estrogen Regulation of Anti-Apoptotic Bcl-2 Family Member Mcl-1 Expression in Breast Cancer Cells

Jennifer L. Schacter et al.

PLOS ONE (2014)

Editorial Material Oncology

BCL-2: A New Therapeutic Target in Estrogen Receptor-Positive Breast Cancer?

Lesley-Ann Martin et al.

CANCER CELL (2013)

Article Medicine, Research & Experimental

ErbB3 downregulation enhances luminal breast tumor response to antiestrogens

Meghan M. Morrison et al.

JOURNAL OF CLINICAL INVESTIGATION (2013)

Article Immunology

Mcl-1 is essential for the survival of plasma cells

Victor Peperzak et al.

NATURE IMMUNOLOGY (2013)

Article Oncology

Pathologic Changes in Breast Cancer After Anti-Estrogen Therapy

Norasate Samarnthai et al.

BREAST JOURNAL (2012)

Article Multidisciplinary Sciences

Sensitization of BCL-2-expressing breast tumors to chemotherapy by the BH3 mimetic ABT-737

Samantha R. Oakes et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2012)

Article Medicine, Research & Experimental

Hyperactivation of phosphatidylinositol-3 kinase promotes escape from hormone dependence in estrogen receptor-positive human breast cancer

Todd W. Miller et al.

JOURNAL OF CLINICAL INVESTIGATION (2010)

Review Cell Biology

Mitochondria and cell death: outer membrane permeabilization and beyond

Stephen W. G. Tait et al.

NATURE REVIEWS MOLECULAR CELL BIOLOGY (2010)

Review Pathology

Nanoparticle-based targeted drug delivery

Rajesh Singh et al.

EXPERIMENTAL AND MOLECULAR PATHOLOGY (2009)

Review Oncology

Biological determinants of endocrine resistance in breast cancer

Elizabeth A. Musgrove et al.

NATURE REVIEWS CANCER (2009)

Article Oncology

ABT-263: A potent and orally bioavailable Bcl-2 family inhibitor

Christin Tse et al.

CANCER RESEARCH (2008)

Article Neurosciences

Mcl-1 is a key regulator of apoptosis during CNS development and after DNA damage

Nicole Arbour et al.

JOURNAL OF NEUROSCIENCE (2008)

Review Biochemistry & Molecular Biology

Mitochondrial outer membrane permeabilization during apoptosis: the innocent bystander scenario

J. E. Chipuk et al.

CELL DEATH AND DIFFERENTIATION (2006)

Review Pharmacology & Pharmacy

Exploiting the enhanced permeability and retention effect for tumor targeting

Arun K. Iyer et al.

DRUG DISCOVERY TODAY (2006)

Article Multidisciplinary Sciences

An inhibitor of Bcl-2 family proteins induces regression of solid tumours

T Oltersdorf et al.

NATURE (2005)

Article Multidisciplinary Sciences

The Bik BH3-only protein is induced in estrogen-starved and antiestrogen-exposed breast cancer cells and provokes apoptosis

JY Hur et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)

Review Biochemistry & Molecular Biology

The hallmarks of cancer

D Hanahan et al.