期刊
CLINICAL KIDNEY JOURNAL
卷 6, 期 1, 页码 1-5出版社
OXFORD UNIV PRESS
DOI: 10.1093/ckj/sft002
关键词
collapsing focal segmental glomerulosclerosis; cytomegalovirus; Epstein-Barr virus; parvovirus B19; podocyte
资金
- NIDDK
- NIH Intramural Research Program
Background. Collapsing glomerulopathy may occur in an idiopathic (primary) form and in association with a wide spectrum of infectious and inflammatory conditions and medications. The association of collapsing glomerulopathy with human immunodeficiency virus (HIV)-1 infection is well established; less certain is the association with other viral infections. Methods. We searched PubMed for articles in all languages that addressed glomerulopathies associated with parvovirus B19, cytomegalovirus (CMV), Epstein-Barr virus (EBV), hepatitis C virus (HCV) and simian virus 40 (SV40). Results. Case reports and small-case series link infection with these common viruses and glomerular injury. The evidence for a pathogenic role is generally stronger for glomerulonephritis than for collapsing glomerulopathy. Conclusions. The evidence linking collapsing glomerulopathy with CMV is relatively strong but not yet conclusive, while the evidence for a pathogenic role for EBV and parvovirus B19 is weaker.
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