期刊
PHYSIOLOGY
卷 33, 期 1, 页码 26-38出版社
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/physiol.00019.2017
关键词
-
类别
资金
- National Institutes of Health (NIH) Ruth L. Kirchstein Postdoctoral Fellowships [AR-071753, AR-065337]
- NIH [AG-034453, AR-060701, AG-049806]
- NIH National Center for Advancing Translational Sciences grant [UL1 TR-000117]
- Ellison Medical Foundation/American Federation for Aging Research Fellowship [EPD 12102]
- NATIONAL INSTITUTE OF ARTHRITIS AND MUSCULOSKELETAL AND SKIN DISEASES [F32AR071753, R01AR060701] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON AGING [R01AG049806] Funding Source: NIH RePORTER
Recent loss-of-function studies show that satellite cell depletion does not promote sarcopenia or unloading-induced atrophy, and does not prevent regrowth. Although overload-induced muscle fiber hypertrophy is normally associated with satellite cell-mediated myonuclear accretion, hypertrophic adaptation proceeds in the absence of satellite cells in fully grown adult mice, but not in young growing mice. Emerging evidence also indicates that satellite cells play an important role in remodeling the extracellular matrix during hypertrophy.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据