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Galectin-3 Binding Protein Secreted by Breast Cancer Cells Inhibits Monocyte-Derived Fibrocyte Differentiation
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Prognostic relevance of LGALS3BP in human colorectal carcinoma
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STRUCTURE (2015)
LGALS3BP, lectin galactoside-binding soluble 3 binding protein, promotes oncogenic cellular events impeded by antibody intervention
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90K, an interferon-stimulated gene product, reduces the infectivity of HIV-1
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A Galectin-3-Dependent Pathway Upregulates Interleukin-6 in the Microenvironment of Human Neuroblastoma
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Cyclophilin C-associated protein regulation of phagocytic functions via NFAT activation in macrophages
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BRAIN RESEARCH (2011)
Dendritic Cell-specific Intercellular Adhesion Molecule 3-grabbing Non-integrin (DC-SIGN) Recognizes a Novel Ligand, Mac-2-binding Protein, Characteristically Expressed on Human Colorectal Carcinomas
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Glycoprotein 90K, downregulated in advanced colorectal cancer tissues, interacts with CD9/CD82 and suppresses the Wnt/β-catenin signal via ISGylation of β-catenin
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BLOOD (2009)
Cyclophilin C-associated protein (CyCAP) knock-out mice spontaneously develop colonic mucosal hyperplasia and exaggerated tumorigenesis after treatment with carcinogen azoxymethane
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High expression of 90K (Mac-2 BP) is associated with poor survival in node-negative breast cancer patients not receiving adjuvant systemic therapies
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INTERNATIONAL JOURNAL OF CANCER (2009)
Leucine-rich Repeats of Bacterial Surface Proteins Serve as Common Pattern Recognition Motifs of Human Scavenger Receptor gp340
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The CD5 ectodomain interacts with conserved fungal cell wall components and protects from zymosan-induced septic shock-like syndrome
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Induction of Mac-2BP by nerve growth factor is regulated by the PI3K/Akt/NF-κB-dependent pathway in the HEK293 cell line
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BMB REPORTS (2008)
Tumor stroma marker endosialin (Tem1) is a binding partner of metastasis-related protein Mac-2 BP/90K
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FASEB JOURNAL (2008)
Serum 90K/Mac-2 Binding Protein (Mac-2BP) as a Response Predictor to Peginterferon and Ribavirin Combined Treatment in Hcv Chronic Patients
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IMMUNOPHARMACOLOGY AND IMMUNOTOXICOLOGY (2008)
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JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Efficient dengue virus (DENV) infection of human muscle satellite cells upregulates type I interferon response genes and differentially modulates MHC I expression on bystander and DENV-infected cells
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JOURNAL OF GENERAL VIROLOGY (2008)
Upstream stimulatory factor regulates constitutive expression and hormonal suppression of the 90K (mac-2BP) protein
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ENDOCRINOLOGY (2007)
Crystal structure of the cysteine-rich domain of scavenger receptor MARCO reveals the presence of a basic and an acidic cluster that both contribute to ligand recognition
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JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Crystal structure of the third extracellular domain of CD5 reveals the fold of a group B scavenger cysteine-rich receptor domain
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JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Cyclophilin C-associated protein is up-regulated during wound healing
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Tumor-associated antigen 90K/Mac-2-binding protein:: Possible role in colon cancer
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JOURNAL OF CELLULAR BIOCHEMISTRY (2006)
Suppression of progression and metastasis of established colon tumors in mice by intravenous delivery of short interfering RNA targeting KITENIN, a metastasis-enhancing protein
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CANCER RESEARCH (2005)
The 90K protein increases major histocompatibility complex class I expression and is regulated by hormones, γ-interferon, and double-strand polynucleotides
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Role of 90K protein in asthma and TH2-type cytokine expression
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Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses
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Functional studies on recombinant domains of Mac-2-binding protein
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JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Variations in serum IL-7 and 90K/Mac-2 binding protein (Mac-2 BP) levels analysed in cohorts of HIV-1 patients and correlated with clinical changes following antiretroviral therapy
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Lack of mother-to-child HIV-1 transmission is associated with elevated serum levels of 90 K immune modulatory protein
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JOURNAL OF CLINICAL IMMUNOLOGY (2000)
Serum protein 90K/Mac-2BP is an independent predictor of disease severity during hepatitis C virus infection
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