期刊
CHEMOSPHERE
卷 207, 期 -, 页码 449-456出版社
PERGAMON-ELSEVIER SCIENCE LTD
DOI: 10.1016/j.chemosphere.2018.05.065
关键词
Toxicity; Triclosan; Chlorine dioxide; Degradation intermediates
资金
- National Natural Science Foundation of China [51378446, 51208468, 51008261]
- Guiding Project of Fujian Province of China [2017Y0079]
- Natural Science Foundation of Fujian Province of China [2017J01491]
- New Century Excellent Talents in Fujian Province University [JA14227]
- Science and Technology Bureau of Xiamen [3502Z20131157, 3502Z20130039]
The mechanism and toxicity of TCS degradation by ClO2 was investigated. Intermediate products during the oxidation process were identified by GC/MS and LC/MS. A microtox bioassay and a SOS/umu assay were employed to evaluate the acute toxicity and genotoxicity of the resulting solutions during the chlorination process. The results showed that the reaction between TCS and ClO2 was of second-order overall. The pseudo first-order rate constants (k(obs)) exhibited significant dependence on solution pH and chlorine dioxide concentration, with the apparent second-order rate constant, k(app), being 7.07 x 10(4) M-1 s(-1) in the pH range of 6.80 -7.02. TCS decomposition was accompanied by the accumulation of 2,4-dichlorophenol (2,4-DCP), and the maximum molar yield ratios of 2,4-DCP/TCS were in the range of 31.71 %-35.43%. The major intermediates identified were 2,7/2,8-dichlorodibenzop-dioxin (2.7/2.8-Cl2DD), 2,4-DCP, 2,4,6-trichlorophenol (2,4,6-TCP), tetraclosan and pentaclosan. The proposed mechanism for TCS oxidation involved the cleavage of the ether link in TCS, chlorination of the phenolic ring and ring closure of a single TCS molecule. The transformation and degradation of TCS led to reduction of the acute toxicity and genotoxicity. However, irregular fluctuations in the toxicity changes indicated that the oxidation of TCS was not a simultaneous detoxification process. (C) 2018 Elsevier Ltd. All rights reserved.
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