期刊
ADVANCED MATERIALS
卷 29, 期 16, 页码 -出版社
WILEY-V C H VERLAG GMBH
DOI: 10.1002/adma.201604932
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资金
- Marie Curie International Incoming Fellowship within the 7th Framework Programme [299936]
- Department of Bioengineering, Imperial College London
- Fight for Sight (UK)
- National Glaucoma Research Program of the BrightFocus Foundation
- National Eye Institute [EY022359]
- European Research Council Consolidator Grant [ERC-2013-CoG 617336 BTVI]
- Rosetrees Trust
- ERC Seventh Framework Programme Consolidator grant Naturale CG [616417]
- Engineering and Physical Sciences Research Council (EPSRC) [EP/K020641/1]
- Wellcome Trust [098411/Z/12/Z]
- EPSRC [EP/K020641/1] Funding Source: UKRI
- Engineering and Physical Sciences Research Council [EP/K020641/1] Funding Source: researchfish
- Fight for Sight [1385/86] Funding Source: researchfish
- Rosetrees Trust [M216] Funding Source: researchfish
Nitric oxide (NO) is able to lower intraocular pressure (IOP); however, its therapeutic effects on outflow physiology are location-and dose-dependent. A NO delivery platform that directly targets the resistance-generating region of the conventional outflow pathway and locally liberates a controlled dose of NO is reported. An increase in outflow facility (decrease in IOP) is demonstrated in a mouse model.
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