期刊
EMERGING INFECTIOUS DISEASES
卷 24, 期 1, 页码 114-117出版社
CENTERS DISEASE CONTROL
DOI: 10.3201/eid2401.171331
关键词
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Postmortem examination results of a patient with Guillain-Barre syndrome and confirmed Zika virus infection revealed demyelination of the sciatic and cranial IV nerves, providing evidence of the acute demyelinating inflammatory polyneuropathy Guillain-Barre syndrome variant. Lack of evidence of Zika virus in nervous tissue suggests that pathophysiology was antibody mediated without neurotropism.
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