期刊
OCULAR SURFACE
卷 15, 期 1, 页码 48-64出版社
ELSEVIER SCIENCE BV
DOI: 10.1016/j.jtos.2016.09.002
关键词
chemical burn; cornea; limbal stem cell deficiency; ocular chemical burn; ocular surface; stem cell
资金
- Clinical Scientist Development Program Award [K12EY021475, R01 EY024349-01A1]
- NEI/NIH [EY01792]
- DoD [MR130543]
- RPB
Chemical injuries frequently result in vision loss, disfigurement, and challenging ocular surface complications. Acute interventions are directed at decreasing the extent of the injury, suppressing inflammation, and promoting ocular surface re-epithelialization. Chronically, management involves controlling inflammation along with rehabilitation and reconstruction of the ocular surface. Future therapies aimed at inhibiting neovascularization and promoting ocular surface regeneration should provide more effective treatment options for the management of ocular chemical injuries.
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