相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Cerebrospinal fluid soluble TREM2 is higher in Alzheimer disease and associated with mutation status
Laura Piccio et al.
ACTA NEUROPATHOLOGICA (2016)
Cerebrospinal fluid sTREM2 levels are associated with gray matter volume increases and reduced diffusivity in early Alzheimer's disease
Juan Domingo Gispert et al.
ALZHEIMERS & DEMENTIA (2016)
sTREM2 cerebrospinal fluid levels are a potential biomarker for microglia activity in early-stage Alzheimer's disease and associate with neuronal injury markers
Marc Suarez-Calvet et al.
EMBO MOLECULAR MEDICINE (2016)
Increased cerebrospinal fluid soluble TREM2 concentration in Alzheimer's disease
Amanda Heslegrave et al.
MOLECULAR NEURODEGENERATION (2016)
TREM2 Lipid Sensing Sustains the Microglial Response in an Alzheimer's Disease Model
Yaming Wang et al.
CELL (2015)
DAP12 Stabilizes the C-terminal Fragment of the Triggering Receptor Expressed on Myeloid Cells-2 (TREM2) and Protects against LPS-induced Pro-inflammatory Response
Li Zhong et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
Apolipoprotein E Is a Ligand for Triggering Receptor Expressed on Myeloid Cells 2 (TREM2)
Yuka Atagi et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
The Triggering Receptor Expressed on Myeloid Cells 2 Binds Apolipoprotein E
Charles C. Bailey et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
TREM-2 promotes macrophage survival and lung disease after respiratory viral infection
Kangyun Wu et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2015)
TREM2 in CNS homeostasis and neurodegenerative disease
Meghan M. Painter et al.
MOLECULAR NEURODEGENERATION (2015)
TREM2 is associated with increased risk for Alzheimer's disease in African Americans
Sheng Chih Jin et al.
MOLECULAR NEURODEGENERATION (2015)
R47H Variant of TREM2 Associated With Alzheimer Disease in a Large Late-Onset Family Clinical, Genetic, and Neuropathological Study
Olena Korvatska et al.
JAMA NEUROLOGY (2015)
Coding variants in TREM2 increase risk for Alzheimer's disease
Sheng Chih Jin et al.
HUMAN MOLECULAR GENETICS (2014)
TL-2 attenuates β-amyloid induced neuronal apoptosis through the AKT/GSK-3β/β-catenin pathway
Xiaolei Zhu et al.
INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY (2014)
Homozygous TREM2 mutation in a family with atypical frontotemporal dementia
Isabelle Le Ber et al.
NEUROBIOLOGY OF AGING (2014)
Heterozygous TREM2 mutations in frontotemporal dementia
Barbara Borroni et al.
NEUROBIOLOGY OF AGING (2014)
TREM2 mutations implicated in neurodegeneration impair cell surface transport and phagocytosis
Gernot Kleinberger et al.
SCIENCE TRANSLATIONAL MEDICINE (2014)
Sequential Proteolytic Processing of the Triggering Receptor Expressed on Myeloid Cells-2 (TREM2) Protein by Ectodomain Shedding and γ-Secretase-dependent Intramembranous Cleavage
Patrick Wunderlich et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2013)
TREM2 and Neurodegenerative Disease
Christiane Reitz et al.
NEW ENGLAND JOURNAL OF MEDICINE (2013)
Variant of TREM2 Associated with the Risk of Alzheimer's Disease
Thorlakur Jonsson et al.
NEW ENGLAND JOURNAL OF MEDICINE (2013)
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro et al.
NEW ENGLAND JOURNAL OF MEDICINE (2013)
Using Exome Sequencing to Reveal Mutations in TREM2 Presenting as a Frontotemporal Dementia-like Syndrome Without Bone Involvement
Rita Joao Guerreiro et al.
JAMA NEUROLOGY (2013)
Nasu-Hakola disease with a splicing mutation of TREM2 in a Japanese family
Y. Numasawa et al.
EUROPEAN JOURNAL OF NEUROLOGY (2011)
Excitotoxicity of TNFα derived from KA activated microglia on hippocampal neurons in vitro and in vivo
Wei Zhu et al.
JOURNAL OF NEUROCHEMISTRY (2010)
Identification of soluble TREM-2 in the cerebrospinal fluid and its association with multiple sclerosis and CNS inflammation
Laura Piccio et al.
BRAIN (2008)
The genetic causes of basal ganglia calcification, dementia, and bone cysts DAP12 and TREM2
HH Klünemann et al.
NEUROLOGY (2005)
The activation of Akt/PKB signaling pathway and cell survival
G Song et al.
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE (2005)
Mutations in two genes encoding different subunits of a receptor signaling complex result in an identical disease phenotype
J Paloneva et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2002)
Cutting edge: Inflammatory responses can be triggered by TREM-1, a novel receptor expressed on neutrophils and monocytes
A Bouchon et al.
JOURNAL OF IMMUNOLOGY (2000)
分享论文
