4.6 Article

Electrochemical study on the fretting corrosion synergistic damage mechanism of Inconel 690 alloy in different dissolved oxygen solution

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ELECTROCHIMICA ACTA
卷 474, 期 -, 页码 -

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PERGAMON-ELSEVIER SCIENCE LTD
DOI: 10.1016/j.electacta.2023.143530

关键词

Fretting corrosion; Synergistic damage mechanism; Electrochemical measurement; Third body layer; Inconel 690 alloy

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In-situ electrochemical measurement was used to investigate the synergistic damage mechanism of fretting corrosion in Inconel 690 alloy under different dissolved oxygen concentrations. The study quantified the synergistic mechanism and found that the minimum damage occurred at a dissolved oxygen concentration of 2 ppm. The influence of the third body layer (TBL) on the fretting corrosion damage mechanism was identified as the key contributor. The formation and rupture of TBL were observed, and it was found that a TBL with excellent ductility could improve contact behavior and reduce fretting corrosion damage, while the breakdown of TBL into hard spinel oxides caused severe abrasive wear and fretting corrosion damage.
In-situ electrochemical measurement was used to investigate the fretting corrosion synergistic damage mecha-nism of Inconel 690 alloy in the solution with different dissolved oxygen (DO) concentrations. The synergistic mechanism of fretting corrosion was quantified. Results show that the minimum fretting corrosion damage of Inconel 690 was obtained at DO concentration of 2 ppm and the corrosion-accelerated wear, which can be represented by the influence of third body layer (TBL) on the fretting corrosion damage mechanism, is the key contributor. The formation and rupture of TBL have been observed under different DO concentrations. In transitional stages of TBL formation and the stage where uniform morphology formed, the TBL with excellent ductility can improve the contact behavior and reduce fretting corrosion damage. In stages during which TBL breaks into hard spinel oxides, severe abrasive wear and fretting corrosion damage will be caused.

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