期刊
CANCER DISCOVERY
卷 6, 期 4, 页码 350-352出版社
AMER ASSOC CANCER RESEARCH
DOI: 10.1158/2159-8290.CD-16-0163
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- NCI NIH HHS [DP2 CA195762] Funding Source: Medline
Cancer genotype-specific synthetic lethal vulnerabilities represent promising therapeutic targets. In this issue of Cancer Discovery, Ogiwara and colleagues uncover a synthetic lethal relationship between two histone acetyl transferase paralogs, CBP and p300, highlighting that cancer cells deficient in CBP are uniquely sensitized to genetic and chemical inhibition of p300. (C) 2016 AACR.
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