期刊
TRENDS IN CELL BIOLOGY
卷 26, 期 8, 页码 624-635出版社
ELSEVIER SCIENCE LONDON
DOI: 10.1016/j.tcb.2016.03.006
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资金
- Biotechnology and Biological Sciences Research Council [BB/K019155/1]
- Cancer Research UK
- Francis Crick Institute
- UK Medical Research Council
- Wellcome Trust
- BBSRC [BBS/E/B/000C0413, BB/K019155/1, BB/H000631/1, BBS/E/B/0000C341] Funding Source: UKRI
- Biotechnology and Biological Sciences Research Council [BBS/E/B/00001221, BB/K019155/1, BBS/E/B/000C0413, BBS/E/B/0000C341, BB/H000631/1] Funding Source: researchfish
- Cancer Research UK [15153] Funding Source: researchfish
- The Francis Crick Institute [10187] Funding Source: researchfish
Autophagy is a catabolic 'self-eating' pathway that is emerging as a crucial integration point in cell physiology. With its own set of genes, the autophagy pathway communicates with virtually all signalling networks and organelles. Recent advances have been made in understanding the origin of the autophagosomal membrane, novel regulators, and the mechanisms by which specific intracellular membranes become autophagy substrates. New studies on non canonical autophagy, mediated by subsets of autophagy proteins, and the role of autophagy proteins in non-autophagy pathways are also emerging in many different biological contexts. Our understanding of canonical autophagy, including membrane origin and autophagy proteins, needs to be considered together with emerging noncanonical pathways.
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