相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Inevitability and containment of replication errors for eukaryotic genome lengths spanning megabase to gigabase
Mohammed Al Mamun et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2016)
Exploring and exploiting the systemic effects of deregulated replication licensing
Theodoros G. Petrakis et al.
SEMINARS IN CANCER BIOLOGY (2016)
Effect of minichromosome maintenance protein 2 deficiency on the locations of DNA replication origins
Dimiter Kunnev et al.
GENOME RESEARCH (2015)
Replication stress activates DNA repair synthesis in mitosis
Sheroy Minocherhomji et al.
NATURE (2015)
The Spatiotemporal Program of DNA Replication Is Associated with Specific Combinations of Chromatin Marks in Human Cells
Franck Picard et al.
PLOS GENETICS (2014)
Bubble-seq analysis of the human genome reveals distinct chromatin-mediated mechanisms for regulating early- and late-firing origins
Larry D. Mesner et al.
GENOME RESEARCH (2013)
PICH: A DNA Translocase Specially Adapted for Processing Anaphase Bridge DNA
Andreas Biebricher et al.
MOLECULAR CELL (2013)
ERCC1 and MUS81-EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis
Valeria Naim et al.
NATURE CELL BIOLOGY (2013)
MUS81 promotes common fragile site expression
Songmin Ying et al.
NATURE CELL BIOLOGY (2013)
Replisome stall events have shaped the distribution of replication origins in the genomes of yeasts
Timothy J. Newman et al.
NUCLEIC ACIDS RESEARCH (2013)
The Subread aligner: fast, accurate and scalable read mapping by seed-and-vote
Yang Liao et al.
NUCLEIC ACIDS RESEARCH (2013)
Unraveling cell type-specific and reprogrammable human replication origin signatures associated with G-quadruplex consensus motifs
Emilie Besnard et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2012)
The BLM helicase contributes to telomere maintenance through processing of late-replicating intermediate structures
Colleen Barefield et al.
NUCLEIC ACIDS RESEARCH (2012)
Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications
Sophie E. Polo et al.
GENES & DEVELOPMENT (2011)
MCM2-7 Form Double Hexamers at Licensed Origins in Xenopus Egg Extract
Agnieszka Gambus et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Replication stress induces 53BP1-containing OPT domains in G1 cells
Jeanine A. Harrigan et al.
JOURNAL OF CELL BIOLOGY (2011)
53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress
Claudia Lukas et al.
NATURE CELL BIOLOGY (2011)
How dormant origins promote complete genome replication
J. Julian Blow et al.
TRENDS IN BIOCHEMICAL SCIENCES (2011)
53BP1 Inhibits Homologous Recombination in Brca1-Deficient Cells by Blocking Resection of DNA Breaks
Samuel F. Bunting et al.
CELL (2010)
Chk1 inhibits replication factory activation but allows dormant origin firing in existing factories
Xin Quan Ge et al.
JOURNAL OF CELL BIOLOGY (2010)
ShortRead: a bioconductor package for input, quality assessment and exploration of high-throughput sequence data
Martin Morgan et al.
BIOINFORMATICS (2009)
Concerted Loading of Mcm2-7 Double Hexamers around DNA during DNA Replication Origin Licensing
Dirk Remus et al.
CELL (2009)
On the origins of ultra-fine anaphase bridges
Kok Lung Chan et al.
CELL CYCLE (2009)
A model for DNA replication showing how dormant origins safeguard against replication fork failure
J. Julian Blow et al.
EMBO REPORTS (2009)
A double-hexameric MCM2-7 complex is loaded onto origin DNA during licensing of eukaryotic DNA replication
Cecile Evrin et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
ReplicationDomain: a visualization tool and comparative database for genome-wide replication timing data
Nodin Weddington et al.
BMC BIOINFORMATICS (2008)
Excess MCM proteins protect human cells from replicative stress by licensing backup origins of replication
Arkaitz Ibarra et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
Dormant origins licensed by excess Mcm2-7 are required for human cells to survive replicative stress
Xin Quan Ge et al.
GENES & DEVELOPMENT (2007)
Replication fork velocities at adjacent replication origins are coordinately modified during DNA replication in human cells
Chiara Conti et al.
MOLECULAR BIOLOGY OF THE CELL (2007)
BLM is required for faithful chromosome segregation and its localization defines a class of ultrafine anaphase bridges
Kok-Lung Chan et al.
EMBO JOURNAL (2007)
Excess Mcm2-7 license dormant origins of replication that can be used under conditions of replicative stress
Anna M. Woodward et al.
JOURNAL OF CELL BIOLOGY (2006)
Tumor suppressor p53 binding protein 1 (53BP1) is involved in DNA damage-signaling pathways
I Rappold et al.
JOURNAL OF CELL BIOLOGY (2001)
Phosphorylation and rapid relocalization of 53BP1 to nuclear foci upon DNA damage
L Anderson et al.
MOLECULAR AND CELLULAR BIOLOGY (2001)