相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。EGCG prevents pressure overload-induced myocardial remodeling by downregulating overexpression of HDAC5 in mice
Xiao Han et al.
INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE (2022)
Upregulation of ERK-EGR1-heparanase axis by HDAC inhibitors provides targets for rational therapeutic intervention in synovial sarcoma
Cinzia Lanzi et al.
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH (2021)
Novel PGC-1α/ATF5 Axis Partly Activates UPRmt and Mediates Cardioprotective Role of Tetrahydrocurcumin in Pathological Cardiac Hypertrophy
Bing Zhang et al.
OXIDATIVE MEDICINE AND CELLULAR LONGEVITY (2020)
Sodium butyrate attenuates angiotensin II-induced cardiac hypertrophy by inhibiting COX2/PGE2 pathway via a HDAC5/HDAC6-dependent mechanism
Linlin Zhang et al.
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE (2019)
Mechanisms of physiological and pathological cardiac hypertrophy
Michinari Nakamura et al.
NATURE REVIEWS CARDIOLOGY (2018)
Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets
Yow Keat Tham et al.
ARCHIVES OF TOXICOLOGY (2015)
HDAC inhibition attenuates cardiac hypertrophy by acetylation and deacetylation of target genes
Jenny Y. Y. Ooi et al.
EPIGENETICS (2015)
EGR1 Functions as a Potent Repressor of MEF2 Transcriptional Activity
Yi Feng et al.
PLOS ONE (2015)
The HDAC Inhibitor LBH589 Induces ERK-Dependent Prometaphase Arrest in Prostate Cancer via HDAC6 Inactivation and Down-Regulation
Mei-Jen Chuang et al.
PLOS ONE (2013)
HDAC-dependent ventricular remodeling
Min Xie et al.
TRENDS IN CARDIOVASCULAR MEDICINE (2013)
Therapeutic Potential for HDAC Inhibitors in the Heart
Timothy A. McKinsey
ANNUAL REVIEW OF PHARMACOLOGY AND TOXICOLOGY, VOL 52 (2012)
Extracellular Signal-Regulated Kinases 1 and 2 Regulate the Balance Between Eccentric and Concentric Cardiac Growth
Izhak Kehat et al.
CIRCULATION RESEARCH (2011)
Histone deacetylase (HDAC) inhibitors attenuate cardiac hypertrophy by suppressing autophagy
Dian J. Cao et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
HDAC-mediated control of ERK- and PI3K-dependent TGF-β-induced extracellular matrix-regulating genes
Matt J. Barter et al.
MATRIX BIOLOGY (2010)
Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice
Haiyun Ling et al.
JOURNAL OF CLINICAL INVESTIGATION (2009)
A new type of ERK1/2 autophosphorylation causes cardiac hypertrophy
Kristina Lorenz et al.
NATURE MEDICINE (2009)
The many roles of histone deacetylases in development and physiology: implications for disease and therapy
Michael Haberland et al.
NATURE REVIEWS GENETICS (2009)
Ca2+/Calmodulin-Dependent protein kinase IIδ and protein kinase D overexpression reinforce the histone deacetylase 5 redistribution in heart failure
Julie Bossuyt et al.
CIRCULATION RESEARCH (2008)
The MEF2D transcription factor mediates stress-dependent cardiac remodeling in mice
Yuri Kim et al.
JOURNAL OF CLINICAL INVESTIGATION (2008)
YY1 Protects Cardiac Myocytes from Pathologic Hypertrophy by Interacting with HDAC5
Carmen C. Sucharov et al.
MOLECULAR BIOLOGY OF THE CELL (2008)
Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3β activity
Chinmay M. Trivedi et al.
NATURE MEDICINE (2007)
CaM kinase II selectively signals to histone deacetylase 4 during cardiornyocyte hypertrophy
Johannes Backs et al.
JOURNAL OF CLINICAL INVESTIGATION (2006)
Early growth response-1 in cardiovascular pathobiology
LM Khachigian
CIRCULATION RESEARCH (2006)
Inhibition of histone deacetylation blocks cardiac hypertrophy induced by angiotensin II infusion and aortic banding
HJ Kee et al.
CIRCULATION (2006)
Egr-1 negatively regulates expression of the sodium-calcium exchanger-1 in cardiomyocytes in vitro and in vivo
CL Wang et al.
CARDIOVASCULAR RESEARCH (2005)
Histone deacetylases 5 and 9 govern responsiveness of the heart to a subset of stress signals and play redundant roles in heart development
SR Chang et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5
RB Vega et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy
CL Zhang et al.
CELL (2002)
Mechanisms of estrogen receptor action in the myocardium -: Rapid gene activation via the ERK1/2 pathway and serum response elements
T de Jager et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
分享论文
