期刊
MOLECULAR CELL
卷 64, 期 6, 页码 1017-1019出版社
CELL PRESS
DOI: 10.1016/j.molcel.2016.11.036
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资金
- NCI NIH HHS [P01 CA174653] Funding Source: Medline
- NIGMS NIH HHS [R01 GM094386, R01 GM041784] Funding Source: Medline
The role of mammalian RAD52 has been mysterious due to the lack of a strong DNA repair phenotype of RAD52-deficient cells. In this issue of Molecular Cell, studies by Bhowmick et al. (2016) and Sotiriou et al. (2016) reveal an unexpected role for RAD52 in promoting DNA synthesis following replication stress.
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