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W. D. Cook et al.
CELL DEATH AND DIFFERENTIATION (2014)
Translocation of mixed lineage kinase domain-like protein to plasma membrane leads to necrotic cell death
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Mixed Lineage Kinase Domain-like Protein MLKL Causes Necrotic Membrane Disruption upon Phosphorylation by RIP3
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MOLECULAR CELL (2014)
Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis
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RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition
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CELL DEATH AND DIFFERENTIATION (2013)
Mlkl knockout mice demonstrate the indispensable role of Mlkl in necroptosis
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CELL RESEARCH (2013)
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The Pseudokinase MLKL Mediates Necroptosis via a Molecular Switch Mechanism
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Two independent pathways of regulated necrosis mediate ischemia-reperfusion injury
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BREAST CANCER RESEARCH AND TREATMENT (2012)
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CELL (2012)
Many stimuli pull the necrotic trigger, an overview
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CELL (2011)
The Adaptor Protein FADD Protects Epidermal Keratinocytes from Necroptosis In Vivo and Prevents Skin Inflammation
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cIAPs Block Ripoptosome Formation, a RIP1/Caspase-8 Containing Intracellular Cell Death Complex Differentially Regulated by cFLIP Isoforms
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Will DNA methylation inhibitors work in solid tumors? A review of the clinical experience with azacitidine and decitabine in solid tumors
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Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation
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CELL (2009)
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MOLECULAR CELL (2007)
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JOURNAL OF CLINICAL ONCOLOGY (2007)
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CANCER RESEARCH (2005)
5-Aza-deoxycytidine induces selective degradation of DNA methyltransferase 1 by a proteasomal pathway that requires the KEN box, bromo-adjacent homology domain, and nuclear localization signal
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MOLECULAR AND CELLULAR BIOLOGY (2005)
Gene expression profiling spares early breast cancer patients from adjuvant therapy: derived and validated in two population-based cohorts
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BREAST CANCER RESEARCH (2005)
Kinase RIP3 is dispensable for normal NF-KBs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and toll-like receptors 2 and 4
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MOLECULAR AND CELLULAR BIOLOGY (2004)
INK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species
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GENES & DEVELOPMENT (2004)
Alkylating DNA damage stimulates a regulated form of necrotic cell death
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GENES & DEVELOPMENT (2004)
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CELL (2003)
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NEW ENGLAND JOURNAL OF MEDICINE (2002)
The RIP-like kinase, RIP3, induces apoptosis and NF-κB nuclear translocation and localizes to mitochondria
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FEBS LETTERS (2000)
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