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Phosphorylation of Tyr1214 within VEGFR-2 triggers the recruitment of Nck and activation of Fyn leading to SAPK2/p38 activation and endothelial cell migration in response to VEGF
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The CD3ε proline-rich sequence, and its interaction with Nck, is not required for T cell development and function
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SLP-76 coordinates Nck-dependent Wiskott-Aldrich syndrome protein recruitment with Vav-1/Cdc42-dependent Wiskott-Aldrich syndrome protein activation at the T cell-APC contact site
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Recruitment of Nck by CD3ε reveals a Ligand-Induced conformational change essential for T cell receptor signaling and synapse formation
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