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NATURE CELL BIOLOGY
卷 24, 期 11, 页码 1568-1570出版社
NATURE PORTFOLIO
DOI: 10.1038/s41556-022-00993-x
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A recent study reveals that the metabolic phosphatase fructose-1,6-bisphosphatase 1 (FBP1) can function as a nuclear protein phosphatase and explains its inhibition mechanism in cancer cells.
In tumours, cancer cells can overcome energy stress via differential regulation of non-canonical 'moonlighting' functions of metabolic enzymes. A study now shows that the metabolic phosphatase fructose-1,6-bisphosphatase 1 (FBP1) can act as a nuclear protein phosphatase and reveals how this process is inhibited in cancer cells.
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