期刊
FUEL
卷 323, 期 -, 页码 -出版社
ELSEVIER SCI LTD
DOI: 10.1016/j.fuel.2022.124283
关键词
Hydrogenated vegetable oil; Biodiesel; NOx emissions; PAHs emissions; Toxicity
资金
- South Coast Air Quality Management District (SCAQMD) [192082]
- California Air Recourses Board CARB [18ISD027]
- NRSA T32 [T32 ES018827]
This study investigates the emissions and potential health effects of using hydrogenated vegetable oil (HVO) and HVO blended with biodiesel as fuels for a legacy off-road diesel engine. The results show that HVO significantly reduces nitrogen oxide (NOx) emissions compared to diesel, while biodiesel blends increase NOx emissions. Both pure HVO and biodiesel blends show reductions in particulate matter (PM) and solid particle numbers. Additionally, the use of biofuels decreases oxidative stress and cytotoxicity in PM emissions.
In this study, the regulated emissions, gaseous toxics, and the physical, chemical, and toxicological properties of particulate matter (PM) emissions from a legacy off-road diesel engine operated on hydrogenated vegetable oil (HVO) and HVO blends with biodiesel were investigated. This is one of the very few studies currently available examining the emissions and potential health effects of HVO and its blends with biodiesel from diesel engines. Extended testing was conducted over the nonroad transient cycle (NRTC) and the 5-mode D2 ISO 8718 cycle. Nitrogen oxide (NOx) emissions showed statistically significant reductions for HVO compared to diesel, whereas the biodiesel blends statistically significant increases in NOx emissions. PM and solid particle number reductions with pure HVO and the biodiesel blends were also observed. Low-molecular weight polycyclic aromatic hydrocarbons (PAHs) were the dominant species in the exhaust for all fuels, with pure HVO and the biodiesel blends showing lower concentrations of these pollutants compared to diesel fuel. Our results showed that the oxidative stress and cytotoxicity in PM emissions decreased with the use of biofuels. Notable correlations were observed between PM emissions and oxidative stress and cytotoxicity, especially elemental carbon and particle-phase PAH emissions.
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