相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。
Review
Biochemistry & Molecular Biology
Korin Sahinyan et al.
Summary: This review discusses the factors and effects of declining numbers and regenerative potential of muscle stem cells (MuSCs) during aging, and emphasizes the importance of understanding these changes for developing effective therapies for age-related muscle defects.
Review
Biochemistry & Molecular Biology
Ilya Potapov et al.
Summary: The stem cell niche is crucial for maintaining genuine quiescent stem cells in adult tissues, with potential deteriorations in extremely old age. Computational tool NicheHotSpotter plays a key role in identifying niche and cell signalling factors for rejuvenating strategies. By analyzing the interaction between stem cells and their microenvironment, novel approaches to enhance stemness and rejuvenate skeletal muscle cells at an advanced age have been discovered.
Review
Biochemistry & Molecular Biology
John F. Bachman et al.
Summary: During development, resident stem cell populations contribute to the growth and maturation of tissue and organs. In skeletal muscle, muscle stem cells play a key role in the maturation of postnatal myofibers. The timing of when these stem cells enter a mature quiescent state and the implications of their loss or dysfunction during development stages are still under debate.
Review
Biochemistry & Molecular Biology
Michael Attwaters et al.
Summary: Skeletal muscle can grow in three ways: by generating new syncytial fibers, adding nuclei from muscle stem cells to existing fibers, or increasing cytoplasmic volume/nucleus. Evidence suggests that the latter two processes contribute to exercise-induced growth. Fiber growth requires an increase in sarcolemmal surface area and cytoplasmic volume at different rates.
Review
Biochemistry & Molecular Biology
Giulio Giuliani et al.
Summary: Fibro/adipogenic progenitor cells (FAPs) play a critical role in modulating muscle regeneration, supporting MuSCs in maintaining tissue homeostasis, but disruption of niche cues can lead to their differentiation into harmful adipocytes and fibroblasts. Efforts are focused on understanding the molecular circuits governing their behavior and utilizing single-cell technologies to study FAP transitional states in muscle regeneration and myopathies.
Article
Biochemistry & Molecular Biology
Yaiza Nunez-Alvarez et al.
Summary: HDAC11 is not essential for adult muscle growth or establishment of the satellite cell population, but its deficiency advances skeletal muscle regeneration after injury. This acceleration is likely due to an enhanced differentiation capacity of satellite cells in the early stages of regeneration in the absence of HDAC11.
Article
Biochemistry & Molecular Biology
Joel R. McDade et al.
Summary: By studying the dynamin-dependent endocytosis and calcium flux, the role of dynamin in wounded skeletal muscle fibers is revealed. Dysferlin deficiency leads to increased FM1-43 dye uptake in muscle fibers, affecting membrane repair mechanisms.
Article
Biochemistry & Molecular Biology
Eduardo de la Vega et al.
Article
Biochemistry & Molecular Biology
Jung Ok Lee et al.
Review
Biochemistry & Molecular Biology
Eric P. Hoffman
Article
Cell Biology
H. Lee Sweeney et al.
COLD SPRING HARBOR PERSPECTIVES IN BIOLOGY
(2018)
Article
Physiology
Christine A. Henderson et al.
COMPREHENSIVE PHYSIOLOGY
(2017)
