4.6 Article

Dexamethasone-Induced Skeletal Muscle Atrophy Increases O-GlcNAcylation in C2C12 Cells

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Physiology

The regulation of muscle mass by endogenous glucocorticoids

Theodore P. Braun et al.

FRONTIERS IN PHYSIOLOGY (2015)

Review Biochemistry & Molecular Biology

Myostatin and the skeletal muscle atrophy and hypertrophy signaling pathways

J. Rodriguez et al.

CELLULAR AND MOLECULAR LIFE SCIENCES (2014)

Review Physiology

O-GlcNAcylation, contractile protein modifications and calcium affinity in skeletal muscle

Caroline Cieniewski-Bernard et al.

FRONTIERS IN PHYSIOLOGY (2014)

Review Biochemistry & Molecular Biology

Glucocorticoid-induced skeletal muscle atrophy

O. Schakman et al.

INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY (2013)

Review Cell Biology

How glucocorticoid receptors modulate the activity of other transcription factors: A scope beyond tethering

Dariusz Ratman et al.

MOLECULAR AND CELLULAR ENDOCRINOLOGY (2013)

Article Cell Biology

Identification of atrogin-1-targeted proteins during the myostatin-induced skeletal muscle wasting

Sudarsanareddy Lokireddy et al.

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2012)

Article Biochemistry & Molecular Biology

O-GlcNAc Transferase Is Involved in Glucocorticoid Receptor-mediated Transrepression

Min-Dian Li et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2012)

Review Geriatrics & Gerontology

Molecular and cellular mechanisms of skeletal muscle atrophy: an update

Alessandro Fanzani et al.

JOURNAL OF CACHEXIA SARCOPENIA AND MUSCLE (2012)

Article Cell Biology

Glycogen synthase kinase-3β is required for the induction of skeletal muscle atrophy

Koen J. P. Verhees et al.

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2011)

Article Cell Biology

Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy

Ping Huang et al.

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2011)

Article Endocrinology & Metabolism

Myoblast models of skeletal muscle hypertrophy and atrophy

Adam P. Sharples et al.

CURRENT OPINION IN CLINICAL NUTRITION AND METABOLIC CARE (2011)

Review Biochemistry & Molecular Biology

O-linked β-N-acetylglucosamine (O-GlcNAc): Extensive crosstalk with phosphorylation to regulate signaling and transcription in response to nutrients and stress

Chutikarn Butkinaree et al.

BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS (2010)

Review Biochemistry & Molecular Biology

Site-specific interplay between O-GlcNAcylation and phosphorylation in cellular regulation

Ping Hu et al.

FEBS LETTERS (2010)

Review Cell Biology

O-GlcNAcylation, an original modulator of contractile activity in striated muscle

C. Cieniewski-Bernard et al.

JOURNAL OF MUSCLE RESEARCH AND CELL MOTILITY (2009)

Article Biochemistry & Molecular Biology

A potent mechanism-inspired O-GlcNAcase inhibitor that blocks phosphorylation of tau in vivo

Scott A. Yuzwa et al.

NATURE CHEMICAL BIOLOGY (2008)

Article Physiology

O-GlcNAc level variations are associated with the development of skeletal muscle atrophy

C Cieniewski-Bernard et al.

JOURNAL OF APPLIED PHYSIOLOGY (2006)

Review Biochemistry & Molecular Biology

Skeletal muscle hypertrophy and atrophy signaling pathways

DJ Glass

INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY (2005)

Article Biochemistry & Molecular Biology

Diabetes and the accompanying hyperglycemia impairs cardiomyocyte calcium cycling through increased nuclear O-GlcNAcylation

RJ Clark et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2003)

Article Biochemical Research Methods

Characterization of a mouse monoclonal antibody specific for O-linked N-acetylglucosamine

FI Comer et al.

ANALYTICAL BIOCHEMISTRY (2001)

Review Biochemistry & Molecular Biology

O-glycosylation of nuclear and cytosolic proteins -: Dynamic interplay between O-GlcNAc and O-phosphate

FI Comer et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2000)