4.2 Article

Bladder Oxidative Stress and HMGB1 Release Contribute to PAR4-Mediated Bladder Pain in Mice

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Medicine, Research & Experimental

Central high mobility group box-1 induces mechanical hypersensitivity with spinal microglial activation in a mouse model of hemi-Parkinson's disease

Fumiaki Sato et al.

Summary: This study investigated the role of central HMGB1 in regulating nociceptive hypersensitivity in a mouse model of PD. The findings suggest that HMGB1 is involved in maintaining nociceptive symptoms in hemi-PD mice via spinal microglial activation, indicating its potential as a therapeutic target for pain associated with PD.

BIOMEDICINE & PHARMACOTHERAPY (2022)

Review Pharmacology & Pharmacy

Role of high-mobility group box 1 and its modulation by thrombomodulin/thrombin axis in neuropathic and inflammatory pain

Ryuichi Tsujita et al.

Summary: HMGB1, a nuclear protein, promotes pain signals and inflammation after being released to the extracellular space. Endogenous HMGB1 accumulated in neurons, perineuronal cells, or immune cells participates in various forms of pain. TM and TMα can reduce HMGB1 degradation, preventing and reversing different types of pathological pain.

BRITISH JOURNAL OF PHARMACOLOGY (2021)

Article Multidisciplinary Sciences

Intravesical CD74 and CXCR4, macrophage migration inhibitory factor (MIF) receptors, mediate bladder pain

Shaojing Ye et al.

Summary: The study showed that urothelial MIF receptors CD74 and CXCR4 mediate bladder pain by releasing urothelial HMGB1. This mechanism may establish persistent pain loops in the bladder, indicating potential novel targets for interrupting bladder pain.

PLOS ONE (2021)

Article Urology & Nephrology

Clinical guidelines for interstitial cystitis/bladder pain syndrome

Yukio Homma et al.

INTERNATIONAL JOURNAL OF UROLOGY (2020)

Review Cell Biology

Evolving complexity of MIF signaling

Stanislovas S. Jankauskas et al.

CELLULAR SIGNALLING (2019)

Review Urology & Nephrology

Phenotyping of interstitial cystitis/bladder pain syndrome

Yoshiyuki Akiyama et al.

INTERNATIONAL JOURNAL OF UROLOGY (2019)

Review Urology & Nephrology

Hunner lesion versus non-Hunner lesion interstitial cystitis/bladder pain syndrome

Kristene E. Whitmore et al.

INTERNATIONAL JOURNAL OF UROLOGY (2019)

Article Multidisciplinary Sciences

Protease-Activated Receptor 4 Induces Bladder Pain through High Mobility Group Box-1

Dimitrios E. Kouzoukas et al.

PLOS ONE (2016)

Article Urology & Nephrology

Diagnosis and Treatment of Interstitial Cystitis/Bladder Pain Syndrome: AUA Guideline Amendment

Philip M. Hanno et al.

JOURNAL OF UROLOGY (2015)

Article Multidisciplinary Sciences

Macrophage Migration Inhibitory Factor Mediates PAR-Induced Bladder Pain

Dimitrios E. Kouzoukas et al.

PLOS ONE (2015)

Article Biochemistry & Molecular Biology

Extracellular High-Mobility Group Box 1 Protein (HMGB1) as a Mediator of Persistent Pain

Nilesh M. Agalave et al.

MOLECULAR MEDICINE (2014)

Meeting Abstract Urology & Nephrology

THE BLADDER AFFERENT RESPONSE TO STIMULATION OF PROTEASE ACTIVATED RECEPTORS (PARS)

D. M. Daly et al.

EUROPEAN UROLOGY SUPPLEMENTS (2011)

Article Cell Biology

Ethyl pyruvate decreases HMGB1 release and ameliorates murine colitis

Shaival H. Dave et al.

JOURNAL OF LEUKOCYTE BIOLOGY (2009)

Article Urology & Nephrology

Voided stain on paper method for analysis of mouse urination

Y. Sugino et al.

NEUROUROLOGY AND URODYNAMICS (2008)

Article Urology & Nephrology

Acute acrolein-induced cystitis in mice

Dale E. Bjorling et al.

BJU INTERNATIONAL (2007)

Article Pharmacology & Pharmacy

FR180204, a novel and selective inhibitor of extracellular signal-regulated kinase, ameliorates collagen-induced arthritis in mice

Makoto Ohori et al.

NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY (2007)