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Inflammatory mediators and the failing heart - Past, present, and the foreseeable future
DL Mann
CIRCULATION RESEARCH (2002)
TNF-α signal transduction in rat neonatal cardiac myocytes:: definition of pathways generating from the TNF-α receptor
G Condorelli et al.
FASEB JOURNAL (2002)
Phosphatidylinositol 3-kinase functionally compartmentalizes the concurrent Gs signaling during β2-adrenergic stimulation
SH Jo et al.
CIRCULATION RESEARCH (2002)
Arachidonic acid mediates dual effect of TNF-α on Ca2+ transients and contraction of adult rat cardiomyocytes
A Amadou et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2002)
Tumor necrosis factor-α in cardiovascular biology and the potential role for anti-tumor necrosis factor-α therapy in heart disease
MN Sack
PHARMACOLOGY & THERAPEUTICS (2002)
Involvement of reactive oxygen species-mediated NF-κB activation in TNF-α-induced cardiomyocyte hypertrophy
Y Higuchi et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2002)
β2-adrenergic receptor agonists increase intracellular free Ca2+ concentration cycling in ventricular cardiomyocytes through p38 and p42/44 MAPK-mediated cytosolic phospholipase A2 activation
S Magne et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Left ventricular remodeling in transgenic mice with cardiac restricted overexpression of tumor necrosis factor
N Sivasubramanian et al.
CIRCULATION (2001)
Dual modulation of cell survival and cell death by β2-adrenergic signaling in adult mouse cardiac myocytes
WZ Zhu et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
The β2-adrenergic receptor delivers an antiapoptotic signal to cardiac myocytes through Gi-dependent coupling to phosphatidylinositol 3′-kinase
A Chesley et al.
CIRCULATION RESEARCH (2000)
Myocardial extracellular matrix remodeling in transgenic mice overexpressing tumor necrosis factor α can be modulated by anti-tumor necrosis factor α therapy
YY Li et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Chronic β-adrenergic stimulation induces myocardial proinflammatory cytokine expression
DR Murray et al.
CIRCULATION (2000)