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Article
Endocrinology & Metabolism
Pandora L. Wander et al.
Summary: This study found that SARS-CoV-2 infection is associated with a higher risk of incident diabetes in men, but not in women, even after accounting for hospital surveillance.
Letter
Endocrinology & Metabolism
Morgan Birabaharan et al.
DIABETES OBESITY & METABOLISM
(2022)
Review
Endocrinology & Metabolism
Rimesh Pal et al.
Summary: COVID-19 affects multiple endocrine organ systems during the disease course. Limited follow-up data suggest that most of the biochemical endocrine dysfunctions observed during acute phase of COVID-19 tend to improve after recovery. New-onset diabetes mellitus has been described post-acute COVID-19, while thyroid/adrenal functions usually normalize on follow-up. Pituitary apoplexy and male hypogonadism have rarely been documented in COVID-19. Routine estimation of serum calcium post-COVID-19 is not warranted.
ENDOCRINE PRACTICE
(2022)
Article
Cell Biology
Verena van der Heide et al.
Summary: In this study, comprehensive single-cell analysis of SARS-CoV-2-infected human pancreatic islets revealed that the infection is strictly dependent on the ACE2 receptor and targets various pancreatic cell types. However, the infection is limited and non-cytopathic, causing only mild cellular perturbations and inflammatory responses. Similar outcomes were observed with endemic coronaviruses. Further evaluation is needed to determine if COVID-19-induced damage to the pancreas and immunological changes increase the risk of diabetes.
Article
Public, Environmental & Occupational Health
Catherine E. Barrett et al.
MMWR-MORBIDITY AND MORTALITY WEEKLY REPORT
(2022)
Article
Pediatrics
Heli Salmi et al.
Summary: A Finnish retrospective cohort study found that more children with T1D had severe DKA at diagnosis during the pandemic due to delays in presentation and diagnosis, rather than a consequence of SARS-CoV-2 infection.
ARCHIVES OF DISEASE IN CHILDHOOD
(2022)
Article
Endocrinology & Metabolism
Wolfgang Rathmann et al.
Summary: This study investigated the incidence of diabetes after infection with Covid-19. The results showed that individuals with Covid-19 had an increased incidence of type 2 diabetes compared to those with acute upper respiratory tract infections (AURI). These findings support the active monitoring of glucose dysregulation after recovery from mild Covid-19.
Article
Endocrinology & Metabolism
Yan Xie et al.
Summary: This study found an increased risk and burden of incident diabetes and antihyperglycaemic use in people with COVID-19 in the post-acute phase compared to a contemporary control group. The risks and burdens of post-acute outcomes were related to the severity of the acute phase of COVID-19.
LANCET DIABETES & ENDOCRINOLOGY
(2022)
Article
Endocrinology & Metabolism
Pandora L. Wander et al.
Summary: This study found that SARS-CoV-2 infection is associated with a higher risk of incident diabetes in men but not in women, even after accounting for hospital surveillance.
Letter
Endocrinology & Metabolism
Thirunavukkarasu Sathish et al.
DIABETES OBESITY & METABOLISM
(2021)
Article
Endocrinology & Metabolism
Kamlesh Khunti et al.
Summary: New-onset diabetes is common among COVID-19 patients and may impact disease severity and outcomes. The underlying mechanisms are complex and require further research. Hospitals should prioritize identification and management of acute hyperglycemia.
Article
Multidisciplinary Sciences
Ziyad Al-Aly et al.
Summary: This study comprehensively describes the post-acute sequelae of COVID-19 using national healthcare databases, finding increased risk of death and use of health resources beyond the first 30 days of illness, as well as incident sequelae in various organ systems, increased use of therapeutic agents, and evidence of laboratory abnormalities. The findings show a risk gradient according to the severity of acute COVID-19 infection and highlight the burden of health loss experienced by patients who survive the acute phase.
Article
Medicine, General & Internal
Daniel Ayoubkhani et al.
Summary: Patients discharged from the hospital after COVID-19 were found to have increased rates of multiorgan dysfunction, with higher risk not only limited to the elderly but also varying across ethnic groups.
BMJ-BRITISH MEDICAL JOURNAL
(2021)
Article
Medicine, General & Internal
Sarah E. Daugherty et al.
Summary: The study evaluated the excess risk and relative hazards for developing incident clinical sequelae after the acute phase of SARS-CoV-2 infection in adults aged 18-65. The results show that individuals infected with SARS-CoV-2 may have a higher risk of developing new clinical sequelae after the acute phase compared to the control groups, with some sequelae more commonly seen in other viral illnesses.
BMJ-BRITISH MEDICAL JOURNAL
(2021)
Article
Endocrinology & Metabolism
Anissa Messaaoui et al.
Summary: The study found that 20% of children developing type 1 diabetes in Belgium during the pandemic tested positive for anti-SARS-CoV-2 antibodies. Children without anti-SARS-CoV-2 antibodies had significantly higher frequencies of insulinoma-associated protein 2 antibodies positivity.
JOURNAL OF PEDIATRIC ENDOCRINOLOGY & METABOLISM
(2021)
Article
Endocrinology & Metabolism
Laura Montefusco et al.
Summary: Patients with COVID-19 may experience hyperglycaemia, insulin resistance, and abnormal glucose metabolism, with these glycaemic abnormalities potentially persisting for at least 2 months after recovery. Further research into metabolic abnormalities in the context of long COVID is warranted.
Editorial Material
Endocrinology & Metabolism
R. Pal et al.
JOURNAL OF ENDOCRINOLOGICAL INVESTIGATION
(2020)
Article
Endocrinology & Metabolism
Paz L. D. Ruiz et al.
Article
Medicine, General & Internal
Florian Krammer et al.
NATURE REVIEWS DISEASE PRIMERS
(2018)
Review
Mathematical & Computational Biology
Claudio Luchini et al.
WORLD JOURNAL OF META-ANALYSIS
(2017)
Editorial Material
Public, Environmental & Occupational Health
Miguel A. Hernan
Review
Medicine, General & Internal
Alessandro Liberati et al.
BMJ-BRITISH MEDICAL JOURNAL
(2009)
