期刊
CANCER CELL
卷 27, 期 4, 页码 435-436出版社
CELL PRESS
DOI: 10.1016/j.ccell.2015.03.014
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资金
- NCI NIH HHS [P30 CA006973] Funding Source: Medline
Recent clinical trials in cancer therapy have demonstrated unprecedented responses through blockade of CTLA-4 and PD-1 immune checkpoint pathways. In a provocative recent paper in Science Translational Medicine, Hatfield and colleagues demonstrate the ability of supplemental oxygen to act as a novel immune checkpoint inhibitor by disrupting the hypoxia-adenosine-A2aR pathway.
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