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PKC-α and TAK-1 are intermediates in the activation of c-Jun NH2-terminal kinase by hypoxia-reoxygenation
Donna P. Frazier et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
Mechanisms of disease - The failing heart - An engine out of fuel
Stefan Neubauer
NEW ENGLAND JOURNAL OF MEDICINE (2007)
The machines that divide and fuse mitochondria
Suzanne Hoppins et al.
ANNUAL REVIEW OF BIOCHEMISTRY (2007)
Mitochondrial dynamics generate equal distribution but patchwork localization of respiratory Complex I
Karin B. Busch et al.
MOLECULAR MEMBRANE BIOLOGY (2006)
OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion
Christian Frezza et al.
CELL (2006)
Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice
S Matsushima et al.
CIRCULATION (2006)
HIF-1-mediated expression of pyruvate dehydrogenase kinase: A metabolic switch required for cellular adaptation to hypoxia
JW Kim et al.
CELL METABOLISM (2006)
Protein kinase A-mediated phosphorylation modulates cytochrome c oxidase function and augments hypoxia and myocardial ischemia-related injury
SK Prabu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Oxidative stress by monoamine oxidase mediates receptor-independent cardiomyocyte apoptosis by serotonin and postischemic myocardial injury
P Bianchi et al.
CIRCULATION (2005)
Decreased contractile and metabolic reserve in peroxisome proliferator-activated receptor-α-null hearts can be rescued by increasing glucose transport and utilization
I Luptak et al.
CIRCULATION (2005)
Targeting an antioxidant to mitochondria decreases cardiac ischemia-reperfusion injury
VJ Adlam et al.
FASEB JOURNAL (2005)
Spl: Regulation of gene expression by phosphorylation
SJ Chu et al.
GENE (2005)
Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion
G Solaini et al.
BIOCHEMICAL JOURNAL (2005)
Overexpression of glutathione peroxidase prevents left ventricular remodeling and failure after myocardial infarction in mice
T Shiomi et al.
CIRCULATION (2004)
Cell-permeable peptide antioxidants targeted to inner mitochondrial membrane inhibit mitochondrial swelling, oxidative cell death, and reperfusion injury
KS Zhao et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Mitochondrial signals initiate the activation of c-Jun N-terminal kinase (JNK) by hypoxia-reoxygenation
CJ Dougherty et al.
FASEB JOURNAL (2004)
Regulation of matrix metalloproteinases by cytokines and reactive oxygen/nitrogen species in the myocardium
DA Siwik et al.
HEART FAILURE REVIEWS (2004)
Endogenously oxidized mitochondrial DNA induces in vivo and in vitro inflammatory responses
LV Collins et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2004)
Synchronized whole cell oscillations in mitochondrial metabolism triggered by a local release of reactive oxygen species in cardiac myocytes
MA Aon et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
The adenine nucleotide translocase: A central component of the mitochondrial permeability transition pore and key player in cell death
AP Halestrap et al.
CURRENT MEDICINAL CHEMISTRY (2003)
Oxidative stress regulates collagen synthesis and matrix metalloproteinase activity in cardiac fibroblasts
DA Siwik et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2001)
Reactive oxygen species (ROS)-induced ROS release: A new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes
DB Zorov et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2000)
Cardiac remodeling-concepts and clinical implications: A consensus paper from an international forum on cardiac remodeling
JN Cohn et al.
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2000)