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Cardiac toxicity from bisphenol A: Are electrophysiology and calcium handling perturbations dose-dependent?

TOXICOLOGY AND APPLIED PHARMACOLOGY (2021)

Cardiac toxicity from bisphenol A exposure in human-induced pluripotent stem cell-derived cardiomyocytes

Sung-Ae Hyun et al.

Summary: Exposure to BPA in human induced pluripotent stem cell-derived cardiomyocytes alters cardiac function, affecting potential, channels, calcium transients, and increasing the risk of arrhythmia.

TOXICOLOGY AND APPLIED PHARMACOLOGY (2021)

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Article Toxicology

Bisphenol S and Bisphenol F Are Less Disruptive to Cardiac Electrophysiology, as Compared With Bisphenol A

Tomas M. Prudencio et al.

Summary: The study demonstrates that BPA and BPF have an immediate inhibitory effect on cardiac ion channels, while BPS is significantly less potent. Further research is needed to fully elucidate the safety profile of bisphenol analogs on the heart.

TOXICOLOGICAL SCIENCES (2021)

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Article Multidisciplinary Sciences

Disruption of neonatal cardiomyocyte physiology following exposure to bisphenol-a

Manelle Ramadan et al.

SCIENTIFIC REPORTS (2018)

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Article Cardiac & Cardiovascular Systems

Physiological response of cardiac tissue to bisphenol a: alterations in ventricular pressure and contractility

Nikki Gillum Posnack et al.

AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2015)

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Cardiac toxicity from bisphenol A: Are electrophysiology and calcium handling perturbations dose-dependent?

相关参考文献

Cardiac toxicity from bisphenol A exposure in human-induced pluripotent stem cell-derived cardiomyocytes

Bisphenol S and Bisphenol F Are Less Disruptive to Cardiac Electrophysiology, as Compared With Bisphenol A

Disruption of neonatal cardiomyocyte physiology following exposure to bisphenol-a

Physiological response of cardiac tissue to bisphenol a: alterations in ventricular pressure and contractility

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