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Stac proteins associate with the critical domain for excitation-contraction coupling in the II-III loop of Ca(V)1.1
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Preconditioning contractions prevent the delayed onset of myofibrillar dysfunction after damaging eccentric contractions
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STAC3 stably interacts through its C1 domain with CaV1.1 in skeletal muscle triads
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Stac3 has a direct role in skeletal muscle-type excitation-contraction coupling that is disrupted by a myopathy-causing mutation
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Three calpain isoforms are autolyzed in rat fast-twitch muscle after eccentric contractions
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