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p63 establishes epithelial enhancers at critical craniofacial development genes
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DREAM and RB cooperate to induce gene repression and cell-cycle arrest in response to p53 activation
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TP63-Mediated Enhancer Reprogramming Drives the Squamous Subtype of Pancreatic Ductal Adenocarcinoma
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CELL REPORTS (2018)
Mutant p63 Affects Epidermal Cell Identity through Rewiring the Enhancer Landscape
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p53 regulates enhancer accessibility and activity in response to DNA damage
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Integration of TP53, DREAM, MMB-FOXM1 and RB-E2F target gene analyses identifies cell cycle gene regulatory networks
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Transcription factor p63 bookmarks and regulates dynamic enhancers during epidermal differentiation
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TP53 engagement with the genome occurs in distinct local chromatin environments via pioneer factor activity
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A novel ATAC-seq approach reveals lineage-specific reinforcement of the open chromatin landscape via cooperation between BAF and p63
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GENES & DEVELOPMENT (2014)
Genome-wide characterization reveals complex interplay between TP53 and TP63 in response to genotoxic stress
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BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2013)
ΔNp63α represses anti-proliferative genes via H2A.Z deposition
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GENES & DEVELOPMENT (2012)
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Genome-wide analysis of p63 binding sites identifies AP-2 factors as co-regulators of epidermal differentiation
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Physical Association of HDAC1 and HDAC2 with p63 Mediates Transcriptional Repression and Tumor Maintenance in Squamous Cell Carcinoma
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p63 regulates Satb1 to control tissue-specific chromatin remodeling during development of the epidermis
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TAp63 induces senescence and suppresses tumorigenesis in vivo
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NATURE CELL BIOLOGY (2009)
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PLOS GENETICS (2009)
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NUCLEIC ACIDS RESEARCH (2008)
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ONCOGENE (2007)
p53 and p73 display common and distinct requirements for sequence specific binding to DNA
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NUCLEIC ACIDS RESEARCH (2007)
p63 protects the female germ line during meiotic arrest
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MOLECULAR CELL (2006)
p63 regulates proliferation and differentiation of developmentally mature keratinocytes
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GENES & DEVELOPMENT (2006)
Both the basal transcriptional activity of the GADD45A gene and its enhancement after ionizing irradiation are mediated by AP-1 element
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p63 heterozygous mutant mice are not prone to spontaneous or chemically induced tumors
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The unique NH2-terminally deleted (ΔN) residues, the PXXP motif, and the PPXY motif are required for the transcriptional activity of the ΔN variant of p63
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TAp63α induces apoptosis by activating signaling via death receptors and mitochondria
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CANCER CELL (2005)
ΔNp63α functions as both a positive and a negative transcriptional regulator and blocks in vitro differentiation of murine keratinocytes
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ONCOGENE (2003)
The ΔNp63α phosphoprotein binds the p21 and 14-3-3σ promoters in vivo and has transcriptional repressor activity that is reduced by Hay-Wells syndrome-derived mutations
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MOLECULAR AND CELLULAR BIOLOGY (2003)
p63 and p73 are required for p53-dependent apoptosis in response to DNA damage
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MOLECULAR CELL (2001)
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MOLECULAR AND CELLULAR BIOLOGY (2001)
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