相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。UCSF ChimeraX: Structure visualization for researchers, educators, and developers
Eric F. Pettersen et al.
PROTEIN SCIENCE (2021)
A common binding motif in the ET domain of BRD3 forms polymorphic structural interfaces with host and viral proteins
Sriram Aiyer et al.
STRUCTURE (2021)
Discovery of RSV-Induced BRD4 Protein Interactions Using Native Immunoprecipitation and Parallel Accumulation-Serial Fragmentation (PASEF) Mass Spectrometry
Morgan Mann et al.
VIRUSES-BASEL (2021)
Highly accurate protein structure prediction with AlphaFold
John Jumper et al.
NATURE (2021)
Epigenetic targeted therapy of stabilized BAP1 in ASXL1 gain-of-function mutated leukemia
Lu Wang et al.
NATURE CANCER (2021)
ASXL3 bridges BRD4 to BAP1 complex and governs enhancer activity in small cell lung cancer
Aileen Patricia Szczepanski et al.
GENOME MEDICINE (2020)
Molecular Regulation of the Polycomb Repressive-Deubiquitinase
Cameron J. Reddington et al.
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES (2020)
Molecular Mechanisms Directing PRC2 Recruitment and H3K27 Methylation
Anne Laugesen et al.
MOLECULAR CELL (2019)
Interactome Rewiring Following Pharmacological Targeting of BET Bromodomains
Jean-Philippe Lambert et al.
MOLECULAR CELL (2019)
The role of ASXL1 in hematopoiesis and myeloid malignancies
Shuhei Asada et al.
CELLULAR AND MOLECULAR LIFE SCIENCES (2019)
The BRD3 ET domain recognizes a short peptide motif through a mechanism that is conserved across chromatin remodelers and transcriptional regulators
Dorothy C. C. Wai et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2018)
Expression of mutant Asxl1 perturbs hematopoiesis and promotes susceptibility to leukemic transformation
Reina Nagase et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2018)
Mutant ASXL1 cooperates with BAP1 to promote myeloid leukaemogenesis
Shuhei Asada et al.
NATURE COMMUNICATIONS (2018)
Efficient proximity labeling in living cells and organisms with TurboID
Tess C. Branon et al.
NATURE BIOTECHNOLOGY (2018)
Targeting Brd4 for cancer therapy: inhibitors and degraders
Yingchao Duan et al.
MEDCHEMCOMM (2018)
A bidentate Polycomb Repressive-Deubiquitinase complex is required for efficient activity on nucleosomes
Martina Foglizzo et al.
NATURE COMMUNICATIONS (2018)
Chromatin regulator Asxl1 loss and Nf1 haploinsufficiency cooperate to accelerate myeloid malignancy
Peng Zhang et al.
JOURNAL OF CLINICAL INVESTIGATION (2018)
Monoubiquitination of ASXLs controls the deubiquitinase activity of the tumor suppressor BAP1
Salima Daou et al.
NATURE COMMUNICATIONS (2018)
Brd4's Bromodomains Mediate Histone H3 Acetylation and Chromatin Remodeling in Pluripotent Cells through P300 and Brgl
Tao Wu et al.
CELL REPORTS (2018)
Gain of function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies
Hui Yang et al.
BLOOD (2018)
The Short Isoform of BRD4 Promotes HIV-1 Latency by Engaging Repressive SWI/SNF Chromatin-Remodeling Complexes
Ryan J. Conrad et al.
MOLECULAR CELL (2017)
ASXL2 is essential for haematopoiesis and acts as a haploinsufficient tumour suppressor in leukemia
Jean-Baptiste Micol et al.
NATURE COMMUNICATIONS (2017)
ASXL3 Is a Novel Pluripotency Factor in Human Respiratory Epithelial Cells and a Potential Therapeutic Target in Small Cell Lung Cancer
Vivek Shukla et al.
CANCER RESEARCH (2017)
Molecular architecture of polycomb repressive complexes
Emily C. Chittock et al.
BIOCHEMICAL SOCIETY TRANSACTIONS (2017)
De novo dominant ASXL3 mutations alter H2A deubiquitination and transcription in Bainbridge-Ropers syndrome
Anshika Srivastava et al.
HUMAN MOLECULAR GENETICS (2016)
De Novo Truncating Variants in ASXL2 Are Associated with a Unique and Recognizable Clinical Phenotype
Vandana Shashi et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2016)
Truncation mutants of ASXL1 observed in myeloid malignancies are expressed at detectable protein levels
Daichi Inoue et al.
EXPERIMENTAL HEMATOLOGY (2016)
BRD4 is a histone acetyltransferase that evicts nucleosomes from chromatin
Ballachanda N. Devaiah et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2016)
Structure of the Brd4 ET domain bound to a C-terminal motif from γ-retroviral integrases reveals a conserved mechanism of interaction
Brandon L. Crowe et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2016)
Structural Mechanism of Transcriptional Regulator NSD3 Recognition by the ET Domain of BRD4
Qiang Zhang et al.
STRUCTURE (2016)
BAP1/ASXL1 recruitment and activation for H2A deubiquitination
Danny D. Sahtoe et al.
NATURE COMMUNICATIONS (2016)
BRD4 is a histone acetyltransferase that evicts nucleosomes from chromatin
Ballachanda N. Devaiah et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2016)
NSD3-Short Is an Adaptor Protein that Couples BRD4 to the CHD8 Chromatin Remodeler
Chen Shen et al.
MOLECULAR CELL (2015)
Cancer-associated ASXL1 mutations may act as gain-of-function mutations of the ASXL1-BAP1 complex
Anand Balasubramani et al.
NATURE COMMUNICATIONS (2015)
Myelodysplastic syndromes are induced by histone methylation-altering ASXL1 mutations
Daichi Inoue et al.
JOURNAL OF CLINICAL INVESTIGATION (2013)
The CRAPome: a contaminant repository for affinity purification-mass spectrometry data
Dattatreya Mellacheruvu et al.
NATURE METHODS (2013)
Integrative Analysis of Complex Cancer Genomics and Clinical Profiles Using the cBioPortal
Jianjiong Gao et al.
SCIENCE SIGNALING (2013)
De novo truncating mutations in ASXL3 are associated with a novel clinical phenotype with similarities to Bohring-Opitz syndrome
Matthew N. Bainbridge et al.
GENOME MEDICINE (2013)
ASXL1 Mutations Promote Myeloid Transformation through Loss of PRC2-Mediated Gene Repression
Omar Abdel-Wahab et al.
CANCER CELL (2012)
Mutations in ASXL1 are associated with poor prognosis across the spectrum of malignant myeloid diseases
Veronique Gelsi-Boyer et al.
JOURNAL OF HEMATOLOGY & ONCOLOGY (2012)
Loss of the Tumor Suppressor BAP1 Causes Myeloid Transformation
Anwesha Dey et al.
SCIENCE (2012)
The Brd4 Extraterminal Domain Confers Transcription Activation Independent of pTEFb by Recruiting Multiple Proteins, Including NSD3
Shaila Rahman et al.
MOLECULAR AND CELLULAR BIOLOGY (2011)
Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia
Mark A. Dawson et al.
NATURE (2011)
De novo nonsense mutations in ASXL1 cause Bohring-Opitz syndrome
Alexander Hoischen et al.
NATURE GENETICS (2011)
The nuclear deubiquitinase BAP1 is commonly inactivated by somatic mutations and 3p21.1 losses in malignant pleural mesothelioma
Matthew Bott et al.
NATURE GENETICS (2011)
Germline mutations in BAP1 predispose to melanocytic tumors
Thomas Wiesner et al.
NATURE GENETICS (2011)
Germline BAP1 mutations predispose to malignant mesothelioma
Joseph R. Testa et al.
NATURE GENETICS (2011)
Histone H2A deubiquitinase activity of the Polycomb repressive complex PR-DUB
Johanna C. Scheuermann et al.
NATURE (2010)
Brd4 Marks Select Genes on Mitotic Chromatin and Directs Postmitotic Transcription
Anup Dey et al.
MOLECULAR BIOLOGY OF THE CELL (2009)
The bromodomain protein Brd4 stimulates G1 gene transcription and promotes progression to S phase
Kazuki Mochizuki et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Brd4 recruits P-TER to chromosomes at late mitosis to promote G1 gene expression and cell cycle progression
Zhiyuan Yang et al.
MOLECULAR AND CELLULAR BIOLOGY (2008)
Semi-supervised learning for peptide identification from shotgun proteomics datasets
Lukas Kall et al.
NATURE METHODS (2007)
Absolute quantification of proteins by LCMSE - A virtue of parallel MS acquisition
JC Silva et al.
MOLECULAR & CELLULAR PROTEOMICS (2006)
Fetal Alz-50 clone 1 (FAC1) protein interacts with the myc-associated zinc finger protein (ZF87/MAZ) and alters its transcriptional activity
KL Jordan-Sciutto et al.
BIOCHEMISTRY (2000)
分享论文
