The study demonstrates the crucial role of PD-L1 in delaying neutrophil apoptosis at the site of inflammation by activating the PI3K-AKT survival pathway.
In this issue of Blood, Wang et al uncover a key role that programmed death ligand 1 (PD-L1) plays in delaying neutrophil apoptosis at the site of inflammation by activation of the phosphatidylinositol 3-kinase (PI3K)-AKT survival pathway.(1)
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