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Rebecca L. Siegel et al.
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F. Sun et al.
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Emma M. Conway et al.
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Adam J. Byrne et al.
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Mingqing Chen et al.
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Zhaoxia Qu et al.
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Jianpeng Sheng et al.
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Fan Sun et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
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Manfred Kopf et al.
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Differential roles of STAT3 in the initiation and growth of lung cancer
J. Zhou et al.
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PDLIM2 expression is driven by vitamin D and is involved in the pro-adhesion, and anti-migration and -invasion activity of vitamin D
E. Vanoirbeek et al.
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Tracy Hussell et al.
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PDLIM2 restricts Th1 and Th17 differentiation and prevents autoimmune disease
Zhaoxia Qu et al.
CELL AND BIOSCIENCE (2012)
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William J. Janssen et al.
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The tumor suppressor gene WWOX links the canonical and noncanonical NF-κB pathways in HTLV-I Tax-mediated tumorigenesis
Jing Fu et al.
BLOOD (2011)
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Rinat Zaynagetdinov et al.
JOURNAL OF IMMUNOLOGY (2011)
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Takashi Tanaka et al.
SCIENCE SIGNALING (2011)
DNA Methylation-Dependent Repression of PDZ-LIM Domain-Containing Protein 2 in Colon Cancer and Its Role as a Potential Therapeutic Target
Zhaoxia Qu et al.
CANCER RESEARCH (2010)
Reactive oxygen species in cancer
Geou-Yarh Liou et al.
FREE RADICAL RESEARCH (2010)
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Zhaoxia Qu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2010)
PDLIM2 suppresses human T-cell leukemia virus type I Tax-mediated tumorigenesis by targeting Tax into the nuclear matrix for proteasomal degradation
Pengrong Yan et al.
BLOOD (2009)
Oxidative stress induced lung cancer and COPD: opportunities for epigenetic therapy
Matthew W. Lawless et al.
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Pengrong Yan et al.
NEOPLASIA (2009)
STATs in cancer inflammation and immunity: a leading role for STAT3
Hua Yu et al.
NATURE REVIEWS CANCER (2009)
Loss of epithelial RelA results in deregulated intestinal proliferative/apoptotic homeostasis and susceptibility to inflammation
Kris A. Steinbrecher et al.
JOURNAL OF IMMUNOLOGY (2008)
PDLIM2-mediated termination of transcription factor NF-κB activation by intranuclear sequestration and degradation of the p65 subunit
Takashi Tanaka et al.
NATURE IMMUNOLOGY (2007)
Endoproteolytic processing of C-terminally truncated NF-κB2 precursors at κB-containing promoters
Guoliang Qing et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)
Stabilization of basally translated NF-κB-inducing kinase (NIK) protein functions as a molecular switch of processing of NF-κB2 p100
GL Qing et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
SLIM is a nuclear ubiquitin E3 ligase that negatively regulates STAT signaling
T Tanaka et al.
IMMUNITY (2005)
Mystique is a new insulin-like growth factor-l-regulated PDZ-LIM domain protein that promotes cell attachment and migration and suppresses anchorage-independent growth
G Loughran et al.
MOLECULAR BIOLOGY OF THE CELL (2005)
Regulation of NF-κB2 p100 processing by its cis-acting domain
GL Qing et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Pdlim2, a novel PDZ-LIM domain protein, interacts with α-actinins and filamin A
M Torrado et al.
INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE (2004)
Tax deregulation of NF-κB2 p100 processing involves both β-TrCP-dependent and -independent mechanisms
ZX Qu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Primary lung tumors in mice as an aid for understanding, preventing, and treating human adenocarcinoma of the lung
AM Malkinson
LUNG CANCER (2001)