相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。Molecular Architecture of a Network of Potential Intracellular EGFR Modulators: ARNO, CaM, Phospholipids, and the Juxtamembrane Segment
Aldino Viegas et al.
STRUCTURE (2020)
Disruption of the EGFR-SQSTM1 interaction by a stapled peptide suppresses lung cancer via activating autophagy and inhibiting EGFR signaling
Jiao-jiao Yu et al.
CANCER LETTERS (2020)
A low-molecular-weight compound exerts anticancer activity against breast and lung cancers by disrupting EGFR/Eps8 complex formation
Meifang Li et al.
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH (2019)
Stapled EGFR peptide reduces inflammatory breast cancer and inhibits additional HER-driven models of cancer
Sabrina A. Maisel et al.
JOURNAL OF TRANSLATIONAL MEDICINE (2019)
Inhibiting Epidermal Growth Factor Receptor Dimerization and Signaling Through Targeted Delivery of a Juxtamembrane Domain Peptide Mimic
Janessa Gerhart et al.
ACS CHEMICAL BIOLOGY (2018)
Cardiac GPCR-Mediated EGFR Transactivation: Impact and Therapeutic Implications
Laurel A. Grisanti et al.
JOURNAL OF CARDIOVASCULAR PHARMACOLOGY (2017)
Temporal and gefitinib-sensitive regulation of cardiac cytokine expression via chronic β-adrenergic receptor stimulation
Laurel A. Grisanti et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2015)
A Structural Perspective on the Regulation of the Epidermal Growth Factor Receptor
Erika Kovacs et al.
ANNUAL REVIEW OF BIOCHEMISTRY, VOL 84 (2015)
β-Adrenergic receptor-mediated transactivation of epidermal growth factor receptor decreases cardiomyocyte apoptosis through differential subcellular activation of ERK1/2 and Akt
Laurel A. Grisanti et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2014)
Conformational Coupling across the Plasma Membrane in Activation of the EGF Receptor
Nicholas F. Endres et al.
CELL (2013)
Inactive ERBB Receptors Cooperate With Reactive Oxygen Species To Suppress Cancer Progression
Matthew R. Hart et al.
MOLECULAR THERAPY (2013)
Regulation of the Ligand-dependent Activation of the Epidermal Growth Factor Receptor by Calmodulin
Hongbing Li et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2012)
A Potential Peptide Therapeutic Derived from the Juxtamembrane Domain of the Epidermal Growth Factor Receptor
Aislyn D. W. Boran et al.
PLOS ONE (2012)
Transactivated EGFR mediates α1-AR-induced STAT3 activation and cardiac hypertrophy
Yan Li et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2011)
G Protein-Dependent and G Protein-Independent Signaling Pathways and Their Impact on Cardiac Function
Douglas G. Tilley
CIRCULATION RESEARCH (2011)
Determination of the Exact Molecular Requirements for Type 1 Angiotensin Receptor Epidermal Growth Factor Receptor Transactivation and Cardiomyocyte Hypertrophy
Nicola J. Smith et al.
HYPERTENSION (2011)
β-Arrestin Mediates β1-Adrenergic Receptor-Epidermal Growth Factor Receptor Interaction and Downstream Signaling
Douglas G. Tilley et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2009)
Novel mode for neutrophil protease cathepsin G-mediated signaling - Membrane shedding of epidermal growth factor is required for cardiomyocyte anoikis (Publication with Expression of Concern. See vol. 132, 2023)
Khadija Rafiq et al.
CIRCULATION RESEARCH (2008)
Quantitative proteomics and phosphoproteomics reveal novel insights into complexity and dynamics of the EGFR signaling network
Sandra Morandell et al.
PROTEOMICS (2008)
b-arrestin-mediated β1-adrenergic receptor transactivation of the EGFR confers cardioprotection
Takahisa Noma et al.
JOURNAL OF CLINICAL INVESTIGATION (2007)
An angiotensin II type 1 receptor mutant lacking epidermal growth factor receptor transactivation does not induce angiotensin II-mediated cardiac hypertrophy
Peiyong Zhai et al.
CIRCULATION RESEARCH (2006)
A small molecule-kinase interaction map for clinical kinase inhibitors
MA Fabian et al.
NATURE BIOTECHNOLOGY (2005)
Genetic alterations that inhibit in vivo pressure-overload hypertrophy prevent cardiac dysfunction despite increased wall stress
G Esposito et al.
CIRCULATION (2002)
Cardiac hypertrophy is not a required compensatory response to short-term pressure overload
JA Hill et al.
CIRCULATION (2000)
分享论文
