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注意：仅列出部分参考文献，下载原文获取全部文献信息。OVOL2-Mediated ZEB1 Downregulation May Prevent Promotion of Actinic Keratosis to Cutaneous Squamous Cell Carcinoma
Maho Murata et al.
JOURNAL OF CLINICAL MEDICINE (2020)
The Transcription Factor OVOL2 Represses ID2 and Drives Differentiation of Trophoblast Stem Cells and Placental Development in Mice
Mariyan J. Jeyarajah et al.
CELLS (2020)
Hypoxia, partial EMT and collective migration: Emerging culprits in metastasis
Kritika Saxena et al.
TRANSLATIONAL ONCOLOGY (2020)
Jarid1b promotes epidermal differentiation by mediating the repression of Ship1 and activation of the AKT/Ovol1 pathway
Xuewei Sun et al.
CELL PROLIFERATION (2019)
OVOL2 induces mesenchymal-to-epithelial transition in fibroblasts and enhances cell-state reprogramming towards epithelial lineages
Kazuhide Watanabe et al.
SCIENTIFIC REPORTS (2019)
Targeting TRPV1 on cellular plasticity regulated by Ovol 2 and Zeb 1 in hepatocellular carcinoma
Chengzhi Xie et al.
BIOMEDICINE & PHARMACOTHERAPY (2019)
Direct Reprogramming Into Corneal Epithelial Cells Using a Transcriptional Network Comprising PAX6, OVOL2, and KLF4
Koji Kitazawa et al.
CORNEA (2019)
Circulating Tumor Cell Clustering Shapes DNA Methylation to Enable Metastasis Seeding
Sofia Gkountela et al.
CELL (2019)
An Ovol2-Zeb1 transcriptional circuit regulates epithelial directional migration and proliferation
Daniel Haensel et al.
EMBO REPORTS (2019)
OVOL1 Influences the Determination and Expansion of iPSC Reprogramming Intermediates
Harunobu Kagawa et al.
STEM CELL REPORTS (2019)
Down-regulation of transcription factor OVOL2 contributes to epithelial-mesenchymal transition in a noninvasive type of trophoblast implantation to the maternal endometrium
Rulan Bai et al.
FASEB JOURNAL (2018)
The role of the OVOL1-OVOL2 axis in normal and diseased human skin
Gaku Tsuji et al.
JOURNAL OF DERMATOLOGICAL SCIENCE (2018)
Ovol2 induces mesenchymal-epithelial transition via targeting ZEB 1 in osteosarcoma
Jijun Liu et al.
ONCOTARGETS AND THERAPY (2018)
OVOL2 links stemness and metastasis via fine-tuning epithelial-mesenchymal transition in nasopharyngeal carcinoma
Xue-Kang Qi et al.
THERANOSTICS (2018)
Interconnected feedback loops among ESRP1, HAS2, and CD44 regulate epithelial-mesenchymal plasticity in cancer
Mohit Kumar Jolly et al.
APL BIOENGINEERING (2018)
Ovol2 gene inhibits the Epithelial-to-Mesenchymal Transition in lung adenocarcinoma by transcriptionally repressing Twist1
Zhi-Hai Wang et al.
GENE (2017)
The GRHL2/ZEB Feedback Loop-A Key Axis in the Regulation of EMT in Breast Cancer
Steven M. Mooney et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2017)
Overexpression of Transcription Factor Ovol2 in Epidermal Progenitor Cells Results in Skin Blistering
Briana Lee et al.
JOURNAL OF INVESTIGATIVE DERMATOLOGY (2017)
Potential role of the OVOL1-OVOL2 axis and c-Myc in the progression of cutaneous squamous cell carcinoma
Takamichi Ito et al.
MODERN PATHOLOGY (2017)
EMT and MET: necessary or permissive for metastasis?
Mohit Kumar Jolly et al.
MOLECULAR ONCOLOGY (2017)
Roles of Grainyhead-like transcription factors in cancer
S. M. Frisch et al.
ONCOGENE (2017)
OVOL2 antagonizes TGF-β signaling to regulate epithelial to mesenchymal transition during mammary tumor metastasis
Rong-Si Wu et al.
ONCOTARGET (2017)
OVOL2, an Inhibitor of WNT Signaling, Reduces Invasive Activities of Human and Mouse Cancer Cells and Is Down-regulated in Human Colorectal Tumors
Guo-Dong Ye et al.
GASTROENTEROLOGY (2016)
Expression of Ovol2 is related to epithelial characteristics and shows a favorable clinical outcome in hepatocellular carcinoma
Hui Fu et al.
ONCOTARGETS AND THERAPY (2016)
Stability of the hybrid epithelial/mesenchymal phenotype
Mohit Kumar Jolly et al.
ONCOTARGET (2016)
OVOL2 Maintains the Transcriptional Program of Human Corneal Epithelium by Suppressing Epithelial-to-Mesenchymal Transition
Koji Kitazawa et al.
CELL REPORTS (2016)
Molecular profiling of tumour budding implicates TGF-mediated epithelial-mesenchymal transition as a therapeutic target in oral squamous cell carcinoma
D. H. Jensen et al.
JOURNAL OF PATHOLOGY (2015)
A Grainyhead-Like 2/Ovo-Like 2 Pathway Regulates Renal Epithelial Barrier Function and Lumen Expansion
Annekatrin Aue et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2015)
OVO-like 1 regulates progenitor cell fate in human trophoblast development
Stephen J. Renaud et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2015)
OVOL guides the epithelial-hybrid-mesenchymal transition
Dongya Jia et al.
ONCOTARGET (2015)
Coupling the modules of EMT and stemness: A tunable 'stemness window' model
Mohit Kumar Jolly et al.
ONCOTARGET (2015)
An Ovol2-Zeb1 Mutual Inhibitory Circuit Governs Bidirectional and Multi-step Transition between Epithelial and Mesenchymal States
Tian Hong et al.
PLOS COMPUTATIONAL BIOLOGY (2015)
Circulating Tumor Cell Clusters Are Oligoclonal Precursors of Breast Cancer Metastasis
Nicola Aceto et al.
CELL (2014)
Mammary Morphogenesis and Regeneration Require the Inhibition of EMT at Terminal End Buds by Ovol2 Transcriptional Repressor
Kazuhide Watanabe et al.
DEVELOPMENTAL CELL (2014)
Transcriptional Mechanisms Link Epithelial Plasticity to Adhesion and Differentiation of Epidermal Progenitor Cells
Briana Lee et al.
DEVELOPMENTAL CELL (2014)
Highly Rapid and Efficient Conversion of Human Fibroblasts to Keratinocyte-Like Cells
Yifang Chen et al.
JOURNAL OF INVESTIGATIVE DERMATOLOGY (2014)
Gene Expression Correlations in Human Cancer Cell Lines Define Molecular Interaction Networks for Epithelial Phenotype
Kurt W. Kohn et al.
PLOS ONE (2014)
The Zinc Finger Transcription Factor Ovol2 Acts Downstream of the Bone Morphogenetic Protein Pathway to Regulate the Cell Fate Decision between Neuroectoderm and Mesendoderm
Ting Zhang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2013)
Transcription Factors OVOL1 and OVOL2 Induce the Mesenchymal to Epithelial Transition in Human Cancer
Hernan Roca et al.
PLOS ONE (2013)
The Cancer Cell Line Encyclopedia enables predictive modelling of anticancer drug sensitivity
Jordi Barretina et al.
NATURE (2012)
Molecular Phylogeny of OVOL Genes Illustrates a Conserved C2H2 Zinc Finger Domain Coupled by Hypervariable Unstructured Regions
Abhishek Kumar et al.
PLOS ONE (2012)
Understanding gene circuits at cell-fate branch points for rational cell reprogramming
Joseph X. Zhou et al.
TRENDS IN GENETICS (2011)
E-cadherin, β-catenin, and ZEB1 in malignant progression of cancer
Otto Schmalhofer et al.
CANCER AND METASTASIS REVIEWS (2009)
Ovol2 Suppresses Cell Cycling and Terminal Differentiation of Keratinocytes by Directly Repressing c-Myc and Notch1
Julie Wells et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2009)
Ovol1 represses its own transcription by competing with transcription activator c-Myb and by recruiting histone deacetylase activity
Mahalakshmi Nair et al.
NUCLEIC ACIDS RESEARCH (2007)
Strain-dependent perinatal lethality of Ovol1-deficient mice and identification of Ovol2 as a downstream target of Ovol1 in skin epidermis
Andy Teng et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2007)
Ovol1 regulates the growth arrest of embryonic epidermal progenitor cells and represses c-myc transcription
M Nair et al.
JOURNAL OF CELL BIOLOGY (2006)
The mouse Ovol2 gene is required for cranial neural tube development
DR Mackay et al.
DEVELOPMENTAL BIOLOGY (2006)
Ovol1 regulates melotic pachytene progression during spermatogenesis by repressing Id2 expression
BA Li et al.
DEVELOPMENT (2005)
Id2 and Id3 define the potency of cell proliferation and differentiation responses to transforming growth factor β and bone morphogenetic protein
M Kowanetz et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
The Ovo/Shavenbaby transcription factor specifies actin remodelling during epidermal differentiation in Drosophila
I Delon et al.
MECHANISMS OF DEVELOPMENT (2003)
Ovol2, a mammalian homolog of Drosophila ovo:: Gene structure, chromosomal mapping, and aberrant expression in blind-sterile mice
B Li et al.
GENOMICS (2002)
The LEF1/β-catenin complex activates movo1, a mouse homolog of Drosophila ovo required for epidermal appendage differentiation
BA Li et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
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