期刊
NEURON
卷 107, 期 2, 页码 219-233出版社
CELL PRESS
DOI: 10.1016/j.neuron.2020.06.032
关键词
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资金
- National Institutes of Health [R011DC016222, U19NS112953]
- Simons Collaboration on the Global Brain
- UCI INP
- NIDCD [K23 DC014747, R01 DC012555, R01 DC017679, R01 DC014253]
- Yale Medical School Fellowship
- National Institute of Nursing Research [1ZIANR000035-01]
- Office of Workforce Diversity, National Institutes of Health
- Rockefeller University Heilbrunn Nurse Scholar Award
- NSF Graduate Research Fellowship
- [R01 DC012383]
- [R21 CA236480]
- NATIONAL INSTITUTE OF NURSING RESEARCH [ZIANR000035] Funding Source: NIH RePORTER
The main neurological manifestation of COVID-19 is loss of smell or taste. The high incidence of smell loss without significant rhinorrhea or nasal congestion suggests that SARS-CoV-2 targets the chemical senses through mechanisms distinct from those used by endemic coronaviruses or other common cold-causing agents. Here we review recently developed hypotheses about how SARS-CoV-2 might alter the cells and cir-cuits involved in chemosensory processing and thereby change perception. Given our limited understanding of SARS-CoV-2 pathogenesis, we propose future experiments to elucidate disease mechanisms and high-light the relevance of this ongoing work to understanding how the virus might alter brain function more broadly.
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