期刊
CLINICAL JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
卷 11, 期 8, 页码 1504-1512出版社
AMER SOC NEPHROLOGY
DOI: 10.2215/CJN.02030216
关键词
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资金
- American Society of Nephrology
- University of Alabama at Birmingham Nephrology Research Center
- University of Alabama at Birmingham Center for Clinical and Translational Science [1UL1TR001417-01]
- National Heart, Lung, and Blood Institute [HL098967]
Vascular access dysfunction remains a major cause of morbidity and mortality in hemodialysis patients. At present there are few effective therapies for this clinical problem. The poor understanding of the pathobiology that leads to arteriovenous fistula (AVF) and graft (AVG) dysfunction remains a critical barrier to development of novel and effective therapies. However, in recent years we have made substantial progress in our understanding of the mechanisms of vascular access dysfunction. This article presents recent advances and new insights into the pathobiology of AVF and AVG dysfunction and highlights potential therapeutic targets to improve vascular access outcomes.
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