期刊
CELL
卷 166, 期 2, 页码 273-274出版社
CELL PRESS
DOI: 10.1016/j.cell.2016.06.054
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资金
- NIEHS NIH HHS [DP1 ES024088] Funding Source: Medline
- NIMH NIH HHS [R01 MH093372] Funding Source: Medline
- NINDS NIH HHS [R01 NS081127] Funding Source: Medline
A study finds that deficits in touch-sensing somatosensory neurons contribute to social interaction and anxiety phenotypes in mouse models of autism and Rett syndrome. These findings suggest that some core symptoms of autism might originate from aberrant development or function of the peripheral nervous system.
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