期刊
NEUROTHERAPEUTICS
卷 17, 期 2, 页码 436-445出版社
SPRINGER
DOI: 10.1007/s13311-019-00829-x
关键词
Subarachnoid hemorrhage; Inflammation; Microglia; Astrocytes; Neutrophils; Delayed cerebral injury
资金
- NINDS NIH HHS [R01 NS074997] Funding Source: Medline
Aneurysmal subarachnoid hemorrhage (SAH) is a devastating disease that leads to poor neurological outcomes and is characterized by both vascular and neural pathologies. Recent evidence demonstrates that inflammation mediates many of the vascular and neural changes observed after SAH. Although most studies focus on inflammatory mediators such as cytokines, the ultimate effectors of inflammation in SAH are parenchymal brain and peripheral immune cells. As such, the present review will summarize our current understanding of the cellular changes of both CNS parenchymal and peripheral immune cells after SAH.
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