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CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY (2009)
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50Hz Extremely Low Frequency Electromagnetic Fields Enhance Protein Carbonyl Groups Content in Cancer Cells: Effects on Proteasomal Systems
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JOURNAL OF NUTRITIONAL BIOCHEMISTRY (2009)
The proteasome: Overview of structure and functions
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PROCEEDINGS OF THE JAPAN ACADEMY SERIES B-PHYSICAL AND BIOLOGICAL SCIENCES (2009)
Epigenetics: Definition, Mechanisms and Clinical Perspective
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SEMINARS IN REPRODUCTIVE MEDICINE (2009)
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Rong Wang et al.
JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY (2008)
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DJ Kuhn et al.
FRONTIERS IN BIOSCIENCE-LANDMARK (2005)
Anti-proliferative and proapoptotic effects of (-)-epigallocatechin-3-gallate on human melanoma: Possible implications for the chemoprevention of melanoma
M Nihal et al.
INTERNATIONAL JOURNAL OF CANCER (2005)
(-)-epigallocatechin gallate and polyphenon E inhibit growth and activation of the epidermal growth factor receptor and human epidermal growth factor receptor-2 signaling pathways in human colon cancer cells
M Shimizu et al.
CLINICAL CANCER RESEARCH (2005)
Green tea constituent epigallocatechin-3-gallate selectively inhibits COX-2 without affecting COX-1 expression in human prostate carcinoma cells
T Hussain et al.
INTERNATIONAL JOURNAL OF CANCER (2005)
EGCG, a major component of green tea, inhibits VEGF production by swine granulosa cells
G Basini et al.
BIOFACTORS (2005)
Regulation of immune cells by local. tissue oxygen tension: Hif1 alpha and adenosine receptors
M Sitkovsky et al.
NATURE REVIEWS IMMUNOLOGY (2005)
A receptor for green tea polyphenol EGCG
H Tachibana et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2004)
Modulation of phosphatidylinositol-3-kinase/protein kinase B- and mitogen-activated protein kinase-pathways by tea polyphenols in human prostate cancer cells
IA Siddiqui et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2004)
MicroRNAs: Genomics, biogenesis, mechanism, and function
DP Bartel
CELL (2004)
Inhibition of the vascular-endothelial growth factor-induced intracellular signaling and mitogenesis of human endothelial cells by epigallocatechin-3 gallate
T Neuhaus et al.
EUROPEAN JOURNAL OF PHARMACOLOGY (2004)
Epigallocatechin-3-gallate, constituent of green tea, suppresses the LPS-induced phenotypic and functional maturation of murine dendritic cells through inhibition of mitogen-activated protein kinases and NF-κB
SC Ahn et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2004)
VEGF receptor phosphorylation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia
YK Lee et al.
BLOOD (2004)
Mechanisms of inhibition of tumor angiogenesis and vascular tumor growth by epigallocatechin-3-gallate
G Fassina et al.
CLINICAL CANCER RESEARCH (2004)
Oral consumption of green tea polyphenols inhibits insulin-like growth factor-i-induced signaling in an autochthonous mouse model of prostate cancer
VM Adhami et al.
CANCER RESEARCH (2004)
A potential prodrug for a green tea polyphenol proteasome inhibitor: evaluation of the peracetate ester of (-)-epigallocatechin gallate [(-)-EGCG]
WH Lam et al.
BIOORGANIC & MEDICINAL CHEMISTRY (2004)
Green tea epigallocatechin-3-gallate mediates T cellular NF-κB inhibition and exerts neuroprotection in autoimmune encephalomyelitis
O Aktas et al.
JOURNAL OF IMMUNOLOGY (2004)
Laminin-induced signaling in tumor cells:: The role of the Mr 67,000 laminin receptor
V Givant-Horwitz et al.
CANCER RESEARCH (2004)
Suppression of UVB-induced phosphorylation of mitogen-activated protein kinases and nuclear factor kappa B by green tea polyphenol in SKH-1 hairless mice
F Afaq et al.
ONCOGENE (2003)
Green tea catechins inhibit VEGF-induced angiogenesis in vitro through suppression of VE-cadherin phosphorylation and inactivation of Akt molecule
FY Tang et al.
INTERNATIONAL JOURNAL OF CANCER (2003)
Epigallocatechin-3-gallate-induced stress signals in HT-29 human colon adenocarcinoma cells
C Chen et al.
CARCINOGENESIS (2003)
Comparative effects of polyphenols from green tea (EGCG) and soybean (genistein) on VEGF and IL-8 release from normal human keratinocytes stimulated with the proinflammatory cytokine TNFα
S Trompezinski et al.
ARCHIVES OF DERMATOLOGICAL RESEARCH (2003)
Inhibition of ultraviolet B-mediated activation of nuclear factor κB in normal human epidermal keratinocytes by green tea Constituent (-)-epigallocatechin-3-gallate
F Afaq et al.
ONCOGENE (2003)
Epimerization of tea catechins and O-methylated derivatives of (-)-epigallocatechin-3-O-gallate:: Relationship between epimerization and chemical structure
M Suzuki et al.
JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY (2003)
Inhibition of phorbol ester-induced COX-2 expression by epigallocatechin gallate in mouse skin and cultured human mammary epithelial cells
JK Kundu et al.
JOURNAL OF NUTRITION (2003)
Exceptionally high protection of photocarcinogenesis by topical application of (-)-epigallocatechin-3-gallate in hydrophilic cream in SKH-1 hairless mouse model: Relationship to inhibition of UVB-induced global DNA hypomethylation
A Mittal et al.
NEOPLASIA (2003)
Green tea polyphenol epigallocatechin-3-gallate inhibits the IL-1β-induced activity and expression of cyclooxygenase-2 and nitric oxide synthase-2 in human chondrocytes
S Ahmed et al.
FREE RADICAL BIOLOGY AND MEDICINE (2002)
Black tea represents a major source of dietary phenolics among regular tea drinkers
AR Rechner et al.
FREE RADICAL RESEARCH (2002)
Comparative antimutagenic and anticlastogenic effects of green tea and black tea: a review
S Gupta et al.
MUTATION RESEARCH-REVIEWS IN MUTATION RESEARCH (2002)
Green tea inhibits vascular endothelial growth factor (VEGF) induction in human breast cancer cells
MR Sartippour et al.
JOURNAL OF NUTRITION (2002)
Synthetic analogs of green tea polyphenols as proteasome inhibitors
DM Smith et al.
MOLECULAR MEDICINE (2002)
Tea catechins inhibit angiogenesis endothelial cell growth, migration in vitro, measured by human and tube formation, through inhibition of VEGF receptor binding
T Kondo et al.
CANCER LETTERS (2002)
Green tea Polyphenol stimulates a Ras, MEKK1, MEK3, and p38 cascade to increase activator protein 1 factor-dependent involucrin gene expression in normal human keratinocytes
S Balasubramanian et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Green tea catechins decrease apolipoprotein B-100 secretion from HepG2 cells
WL Yee et al.
MOLECULAR AND CELLULAR BIOCHEMISTRY (2002)
Inhibition of UVB-induced oxidative stress-mediated phosphorylation of mitogen-activated protein kinase signaling pathways in cultured human epidermal keratinocytes by green tea polyphenol (-)-epigallocatechin-3-gallate
SK Katiyar et al.
TOXICOLOGY AND APPLIED PHARMACOLOGY (2001)
Ester bond-containing tea polyphenols potently inhibit proteasome activity in vitro and in vivo
S Nam et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells
YD Jung et al.
BRITISH JOURNAL OF CANCER (2001)
Green tea and its catechins inhibit breast cancer xenografts
MR Sartippour et al.
NUTRITION AND CANCER-AN INTERNATIONAL JOURNAL (2001)
Activation of antioxidant-response element (ARE), mitogen-activated protein kinases (MAPKs) and caspases by major green tea polyphenol components during cell survival and death
C Chen et al.
ARCHIVES OF PHARMACAL RESEARCH (2000)
Quantitative analysis of flavan-3-ols in Spanish foodstuffs and beverages
S de Pascual-Teresa et al.
JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY (2000)
Comparative analysis of tea catechins and theaflavins by highperformance liquid chromatography and capillary electrophoresis
BL Lee et al.
JOURNAL OF CHROMATOGRAPHY A (2000)
Growth inhibition, cell-cycle dysregulation, and induction of apoptosis by green tea constituent (-)-epigallocatechin-3-gallate in androgen-sensitive and androgen-insensitive human prostate carcinoma cells
S Gupta et al.
TOXICOLOGY AND APPLIED PHARMACOLOGY (2000)
Suppression of azoxymethane-induced preneoplastic lesions and inhibition of cyclooxygenase-2 activity in the colonic mucosa of rats drinking a crude green tea extract
N Metz et al.
NUTRITION AND CANCER-AN INTERNATIONAL JOURNAL (2000)