Sonolytic degradation of bisphenol S: Effect of dissolved oxygen and peroxydisulfate, oxidation products and acute toxicity

In this paper, the kinetics of bisphenol S (BPS) degradation in the presence of peroxydisulfate (PDS) or dissolved oxygen (DO) in ultrasound (US) system were investigated. For PDS (US/PDS), increased PDS concentration result in faster BPS degradation, but the enhancement was not remarkable with multiplying PDS dosages. Therefore, heterogeneous PDS activation model based on a Langmuir-type adsorption mechanism was proposed to explain the trait of BPS abatement. The equilibrium constant of PDS (K-pDs) was calculated to be 2.91 x 10(-4)/mu M, which was much lower than that of BPS, suggesting that PDS was hard to adsorb on the gas-liquid interface of the cavitation bubble following by activation. Besides, the formation of center dot OH and SO4 center dot- in US/PDS system was reinvestigated. The result showed that SO4 center dot- rather than center dot OH was the predominant radical, which was quite different from previous study. Dissolved oxygen largely improve the degradation of BPS in US system and center dot OH rather than Or was proved to be the main reactive oxygen species (ROS). The improvement of center dot OH generation possibly caused by the reaction of DO with center dot H so that it cannot recombine with center dot OH. The transformation of the BPS in US system mainly included BPS radical polymerization, hydroxylation and hydrolysis. Frustratingly, the acute toxicity assay of Vibrio fischeri suggests that the degradation products of BPS are more toxic. These results will improve the understanding on the activation mechanisms of PDS and the role of dissolved oxygen play in US. Further investigations may need to explore other treatment ways of BPS and evaluate the acute toxicity of degradation products. (C) 2019 Elsevier Ltd. All rights reserved: