相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Genome-wide discovery of somatic coding and noncoding mutations in pediatric endemic and sporadic Burkitt lymphoma
Bruno M. Grande et al.
BLOOD (2019)
ROS signalling in the biology of cancer
Jennifer N. Moloney et al.
SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY (2018)
The DNA damage response pathway in normal hematopoiesis and malignancies
Domenico Delia et al.
INTERNATIONAL JOURNAL OF HEMATOLOGY (2017)
Tumour virus epidemiology
Ruth M. Lunn et al.
PHILOSOPHICAL TRANSACTIONS OF THE ROYAL SOCIETY B-BIOLOGICAL SCIENCES (2017)
Regulation of Telomere Homeostasis during Epstein-Barr virus Infection and Immortalization
Siamak A. Kamranvar et al.
VIRUSES-BASEL (2017)
MTH1 as a Chemotherapeutic Target: The Elephant in the Room
Govindi J. Samaranayake et al.
CANCERS (2017)
Not breathing is not an option: How to deal with oxidative DNA damage
Enni Markkanen
DNA REPAIR (2017)
Oxidative stress enables Epstein-Barr virus-induced B-cell transformation by posttranscriptional regulation of viral and cellular growth-promoting factors
X. Chen et al.
ONCOGENE (2016)
Hypoxic Signaling and the Cellular Redox Tumor Environment Determine Sensitivity to MTH1 Inhibition
Lars Brautigam et al.
CANCER RESEARCH (2016)
Reactive Oxygen Species Dictate the Apoptotic Response of Melanoma Cells to TH588
Jia Yu Wang et al.
JOURNAL OF INVESTIGATIVE DERMATOLOGY (2016)
Potent and Selective Inhibitors of MTH1 Probe Its Role in Cancer Cell Survival
Jason G. Kettle et al.
JOURNAL OF MEDICINAL CHEMISTRY (2016)
Base Excision Repair, a Pathway Regulated by Posttranslational Modifications
Rachel J. Carter et al.
MOLECULAR AND CELLULAR BIOLOGY (2016)
Proteomic profiling of small-molecule inhibitors reveals dispensability of MTH1 for cancer cell survival
Tatsuro Kawamura et al.
SCIENTIFIC REPORTS (2016)
Glutathione and Thioredoxin Antioxidant Pathways Synergize to Drive Cancer Initiation and Progression
Isaac S. Harris et al.
CANCER CELL (2015)
Oncogene-induced reactive oxygen species fuel hyperproliferation and DNA damage response activation
M. Ogrunc et al.
CELL DEATH AND DIFFERENTIATION (2014)
Cellular Levels of 8-Oxoguanine in either DNA or the Nucleotide Pool Play Pivotal Roles in Carcinogenesis and Survival of Cancer Cells
Yusaku Nakabeppu
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES (2014)
Stereospecific targeting of MTH1 by (S)-crizotinib as an anticancer strategy
Kilian V. M. Huber et al.
NATURE (2014)
MTH1 inhibition eradicates cancer by preventing sanitation of the dNTP pool
Helge Gad et al.
NATURE (2014)
Tumor viruses and replicative immortality - Avoiding the telomere hurdle
Xinsong Chen et al.
SEMINARS IN CANCER BIOLOGY (2014)
Association of DNA Base-excision Repair XRCC1, OGG1 and APE1 Gene Polymorphisms with Nasopharyngeal Carcinoma Susceptibility in a Chinese Population
Qing Li et al.
ASIAN PACIFIC JOURNAL OF CANCER PREVENTION (2013)
Oxidative Stress and DNA Damage in Human Gastric Carcinoma: 8-Oxo-7′8-dihydro-2′-deoxyguanosine (8-oxo-dG) as a Possible Tumor Marker
Silvia Borrego et al.
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES (2013)
Reactive Oxygen Species Are Induced by Kaposi's Sarcoma-Associated Herpesvirus Early during Primary Infection of Endothelial Cells To Promote Virus Entry
Virginie Bottero et al.
JOURNAL OF VIROLOGY (2013)
The Epstein-Barr virus nuclear antigen-1 reprograms transcription by mimicry of high mobility group A proteins
Giuseppe Coppotelli et al.
NUCLEIC ACIDS RESEARCH (2013)
HCV and Oxidative Stress in the Liver
Alexander V. Ivanov et al.
VIRUSES-BASEL (2013)
An ATM/Chk2-Mediated DNA Damage-Responsive Signaling Pathway Suppresses Epstein-Barr Virus Transformation of Primary Human B Cells
Pavel A. Nikitin et al.
CELL HOST & MICROBE (2010)
HTLV-1 Tax: Linking transformation, DNA damage and apoptotic T-cell death
Katerina Chlichlia et al.
CHEMICO-BIOLOGICAL INTERACTIONS (2010)
Transcription Profiling of Epstein-Barr Virus Nuclear Antigen (EBNA)-1 Expressing Cells Suggests Targeting of Chromatin Remodeling Complexes
Ramakrishna Sompallae et al.
PLOS ONE (2010)
Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints
B. Gruhne et al.
ONCOGENE (2009)
The Epstein-Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species
Bettina Gruhne et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
EBV and genomic instability-A new look at the role of the virus in the pathogenesis of Burkitt's lymphoma
Bettina Gruhne et al.
SEMINARS IN CANCER BIOLOGY (2009)
An oncogene-induced DNA damage model for cancer development
Thanos D. Halazonetis et al.
SCIENCE (2008)
Epstein-Barr virus promotes genomic instability in Burkitt's lymphoma
S. A. Kamranvar et al.
ONCOGENE (2007)
Inhibition of heavy chain and β2-microglobulin synthesis as a mechanism of major histocompatibility complex class I downregulation during Epstein-Barr virus replication
Andre Ortlieb Guerreiro-Cacais et al.
JOURNAL OF VIROLOGY (2007)
Reactive oxygen signaling and MAPK activation distinguish Epstein-Barr virus (EBV)-positive versus EBV-negative Burkitt's lymphoma
F Cerimele et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growth
MS Kang et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
Epstein-Barr virus gH is essential for penetration of B cells but also plays a role in attachment of virus to epithelial cells
SJ Molesworth et al.
JOURNAL OF VIROLOGY (2000)